|1.||Dörner, Andrea: 5 articles (01/2016 - 01/2006)|
|2.||Schultheiss, Heinz-Peter: 5 articles (01/2016 - 01/2006)|
|3.||Jang, Ji-Young: 3 articles (01/2013 - 01/2008)|
|4.||Kim, Chul-Woo: 3 articles (01/2013 - 01/2008)|
|5.||Jeon, Yoon-Kyung: 3 articles (01/2013 - 01/2008)|
|6.||Miura, Tetsuji: 3 articles (11/2010 - 07/2009)|
|7.||Chen, Xin Jie: 3 articles (12/2008 - 08/2002)|
|8.||Yuan, Jing: 3 articles (08/2008 - 07/2005)|
|9.||Winter, Julia: 2 articles (01/2016 - 04/2014)|
|10.||Klumpe, Inga: 2 articles (01/2016 - 04/2014)|
|2.||Dilated Cardiomyopathy (Cardiomyopathy, Congestive)
01/01/2011 - "Adenine nucleotide translocase 1 deficiency results in dilated cardiomyopathy with defects in myocardial mechanics, histopathological alterations, and activation of apoptosis."
01/01/2006 - "An isoform shift in the cardiac adenine nucleotide translocase expression alters the kinetic properties of the carrier in dilated cardiomyopathy."
05/01/2002 - "Mitochondrial adenine nucleotide translocase (ANT) is a specific target for the autoantibody response in idiopathic dilated cardiomyopathy (IDCM). "
02/01/1995 - "The presence and dynamic change of autoantibody against ADP/ATP carrier in patients with dilated cardiomyopathy (DCM) was studied by using indirect micro-solid-phase radioimmunoassay. "
02/01/1995 - "[Dynamic observation and significance of autoantibody against ADP/ATP carrier in dilated cardiomyopathy]."
06/01/1992 - "After 30 minutes of regional ischemia, postischemic restoration of the mitochondrial AdN pool occurs between 1 and 7 days; this restoration is preceded by recovery of respiratory and adenine nucleotide translocase functions. "
07/01/1989 - "Five minutes of total global ischemia without reperfusion produced a significant depression in adenine nucleotide translocase in subsarcolemmal mitochondria (SLM), whereas a minimum of 10 min ischemia was required to observe a significant depression in interfibrillar mitochondria (IFM). "
05/01/1984 - "Therefore, the effects of ischemia on adenine nucleotide translocase activity could not be established. "
01/01/1975 - "Role of adenine nucleotide translocase in metabolic change caused by ischemia."
05/01/2001 - "This study investigates the effect of ischemic preconditioning on mitochondrial function, including functional coupling between the adenine nucleotide translocase and mitochondrial creatine kinase, which is among the first reactions to be altered in ischemia. "
01/01/2016 - "Adenine nucleotide translocase 1 overexpression protects cardiomyocytes against hypoxia via increased ERK1/2 and AKT activation."
05/01/2007 - "Alleviation of fatty acid and hypoxia-reoxygenation-induced proximal tubule deenergization by ADP/ATP carrier inhibition and glutamate."
01/01/2016 - "In this regard, we analysed the effect of adenine nucleotide translocase 1 (ANT1), which facilitates the exchange of ADP and ATP across the mitochondrial membrane, on cell-protective survival signalling under hypoxia. "
01/01/1987 - "In low-flow anoxia fatty acids enhanced the depression of oxidative phosphorylation, the loss of cytochromes, the inhibition of adenine nucleotide translocase and the reduction of mitochondrial Ca2+ sequestration. "
05/01/2007 - "To assess the mechanism for this behavior, we have compared the efficacies of different NEFA for inducing mitochondrial deenergization in permeabilized tubules measured using safranin O uptake and studied the modification of NEFA-induced deenergization by inhibitors of the ADP/ATP carrier and glutamate using both normoxic tubules treated with exogenous NEFA and tubules deenergized during hypoxia-reoxygenation (H/R). "
|5.||Breast Neoplasms (Breast Cancer)
01/01/2010 - "In this study, we examined the question of whether or not adenine nucleotide translocase 2 (ANT2) short hairpin RNA (shRNA)-mediated down-regulation of HER2/neu and inhibitory effects on the PI3K/Akt signaling pathway suppressed migration and invasiveness of breast cancer cells. "
03/01/2012 - "These data should stimulate further research into both the role of the mevalonate pathway and the accumulation of pyrophosphate compounds like ApppI in tumorigenesis and differentiation and their potential apart from the inhibition of mitochondrial ADP/ATP translocase and apoptosis, since such effects might well be responsible for the adjuvant ZA treatment benefit of patients suffering from breast cancer."
|1.||Adenosine Diphosphate (ADP)
|2.||Proteins (Proteins, Gene)
|4.||Electron Transport Complex IV (Cytochrome c Oxidase)
|7.||Small Interfering RNA (siRNA)
|9.||Mitochondrial Form Creatine Kinase