|1.||Sahai, Erik: 5 articles (01/2016 - 08/2006)|
|2.||Sanz-Moreno, Victoria: 4 articles (01/2016 - 08/2011)|
|3.||Baum, Jake: 4 articles (01/2015 - 03/2008)|
|4.||Paluch, Ewa: 4 articles (09/2012 - 01/2006)|
|5.||Orgaz, Jose L: 3 articles (01/2016 - 01/2014)|
|6.||Angrisano, Fiona: 3 articles (01/2015 - 03/2012)|
|7.||Gaggioli, Cedric: 3 articles (06/2014 - 08/2011)|
|8.||Albrengues, Jean: 3 articles (06/2014 - 08/2011)|
|9.||Charras, Guillaume: 3 articles (06/2013 - 09/2008)|
|10.||Hooper, Steven: 3 articles (06/2013 - 01/2011)|
11/10/2009 - "The perinuclear actin cap is specifically disorganized or eliminated by inhibition of actomyosin contractility and rupture of the LINC complexes, which connect the nucleus to the actin cap. "
05/04/2012 - "The one-step master equation can be used also for efficient computer simulations with linear elastic external load and reveals the sequence of buildup of force and ensemble rupture that is characteristic for reconstituted actomyosin contractility."
09/01/2008 - "They are the result of actomyosin contractions of the cortex, which cause either transient detachment of the cell membrane from the actin cortex or a rupture in the actin cortex. "
05/01/2007 - "Transition into the activated adhesion mode and the increase of the smallest rupture forces were both blocked by inhibitors of actomyosin contractility. "
01/01/2006 - "By analyzing the ruptures thus produced in various myofibril preparations, it was found that the sarcomere structure of myofibrils is maintained by numerous molecular components having the mechanical strength sufficient to sustain the contractile force produced by the actomyosin system. "
|2.||Pulmonary Fibrosis (Hamman Rich Syndrome)
01/01/2007 - "This report summarizes our experience with the protective effects of L 158,809, an angiotensin II (ANG II) receptor blocker, and two angiotensin converting enzyme (ACE) inhibitors in the development of IPS and the role of transforming growth factor beta (TGF-beta) and of alpha-actomyosin (alpha SMA) in pathogenesis of radiation induced pulmonary fibrosis in an experimental model of bone marrow transplant (BMT). "
|3.||Wounds and Injuries (Trauma)
12/01/2013 - "Previous studies have found that an actomyosin ring (purse string) forms around the wound perimeter and contracts to close the wound over the course of several dozens of minutes. "
03/01/2002 - "Recent studies suggested that an actomyosin purse string is crucial for the closure of wounds in single cells. "
11/17/2015 - "Additionally, this signaling leads to the shutdown of actomyosin stress fibers in these same epidermal cells, which may act to release tension within the wound monolayer. "
08/31/2015 - "Actomyosin cable formation is accompanied by junctional rearrangements at the wound margin. "
08/31/2015 - "A supracellular actomyosin cable around the wound coordinates cellular movements and promotes wound closure. "
11/01/1959 - "Studies of actomyosin from cardiac muscle of dogs with experimental congestive heart failure."
09/01/2005 - "Therefore, the replacement of alpha-MHC by beta-MHC (possessing slower actomyosin enzymatic kinetics) may underlie to a significant degree the reduced myocardial shortening velocity and reduced relaxation function in human heart failure. "
11/01/2001 - "Hence, actin hypothesis of reduced contractile activity of myocardial contractile protein system in acute heart failure transforms into the actomyosin concept in chronic heart failure."
05/28/1999 - "Thus, this study provides evidence that adenylate kinase facilitates the transfer of high-energy phosphoryls and signal communication between mitochondria and actomyosin in cardiac muscle, with an increased contribution to cellular phosphotransfer in heart failure. "
08/01/1985 - "It was shown that, in the control hybrid actomyosin, the substitution of normal Straub actin by that from the failing heart resulted in decrease of both the rate and extent of SP. The conclusion was made that both changes in myosin properties and Straub actin underlie the reduced contractility of the myofibrillar protein system in acute and congestive heart failure."
11/16/2015 - "Cell migration underlies metastatic dissemination of cancer cells, and fast "amoeboid" migration in the invasive fronts of tumors is controlled by high levels of actomyosin contractility. "
04/01/2015 - "Actomyosin tension as a determinant of metastatic cancer mechanical tropism."
01/01/2015 - "In this article, we provide a detailed description of the actomyosin cytoskeleton organization during oncogenic extrusion and discuss the implications of cell extrusion in cancer. "
01/01/2015 - "Invadopodia activity has been shown to be dependent on ECM rigidity and cancer cell contractile forces suggesting that rigidity signals can regulate these subcellular structures through actomyosin contractility. "
11/01/2014 - "We find that the identity of engulfing ("winner") and engulfed ("loser") cells is dictated by mechanical deformability controlled by RhoA and actomyosin, where tumor cells with high deformability preferentially engulf and outcompete neighboring cells with low deformability in heterogeneous populations. "
|1.||Transforming Growth Factor beta (TGF-beta)
|2.||Peptidyl-Dipeptidase A (Angiotensin Converting Enzyme)
|3.||Angiotensin-Converting Enzyme Inhibitors (ACE Inhibitors)
|6.||Proteins (Proteins, Gene)
|7.||Adenosine Diphosphate (ADP)
|4.||Protein-Restricted Diet (Diet, Protein Restricted)