|1.||Karsenty, G: 3 articles (07/2005 - 01/2001)|
|2.||Elefteriou, Florent: 2 articles (06/2015 - 09/2003)|
|3.||Shim, Jae-Hyuck: 2 articles (09/2013 - 05/2012)|
|4.||Glimcher, Laurie H: 2 articles (09/2013 - 05/2012)|
|5.||Wein, Marc N: 2 articles (09/2013 - 05/2012)|
|6.||Jones, Dallas C: 2 articles (09/2013 - 05/2012)|
|7.||Karsenty, Gerard: 2 articles (08/2011 - 09/2003)|
|8.||Dacquin, Romain: 2 articles (01/2011 - 02/2004)|
|9.||de Vernejoul, Marie-Christine: 2 articles (03/2010 - 03/2008)|
|10.||Schilling, Arndt F: 2 articles (12/2006 - 02/2005)|
06/01/2013 - "We will also review the mild skeletal dysplasia associated with the currently unexplained high bone mass phenotype and discuss recent advances in osteoporosis therapies arising from improved understanding of rare inherited high BMD disorders."
10/01/2015 - "The impairment of bone remodeling may lead to many skeletal diseases, such as high bone mass syndrome or osteoporosis. "
04/01/2014 - "Inactivation of Vhl in osteochondral progenitor cells causes high bone mass phenotype and protects against age-related bone loss in adult mice."
09/01/2013 - "Inducible knockdown of Shn3 in adult mice resulted in a high-bone mass phenotype, providing evidence that transient blockade of these pathways in adults holds promise as a therapy for osteoporosis."
12/01/2008 - "These results, consistent with the dominant nature of the high bone mass phenotype and normal heart function of Adrbeta2R-deficient mice, suggest that low doses of beta-blockers may have a therapeutic utility in the treatment of osteoporosis with high selectivity for bone tissues."
|2.||Hypertension (High Blood Pressure)
06/01/2015 - "In this study, we show that bilateral vestibular lesions in mice cause a low bone mass phenotype associated with decreased bone formation and increased bone resorption. "
12/01/2015 - "Taken together, these data suggest that AREG overexpression in osteoblasts induces a transient high bone mass phenotype in the trabecular compartment of the appendicular skeleton by a growth-related, non-cell autonomous mechanism, leading to a positive bone balance with unchanged bone formation and lowered bone resorption. "
12/01/2013 - "Osteoclast-specific p130Cas conditional knockout (p130Cas(ΔOCL-) ) mice exhibit a high bone mass phenotype caused by defect in multinucleation and cytoskeleton organization causing bone resorption deficiency. "
05/22/2012 - "Finally, selective deletion of Shn3 in the mesenchymal lineage recapitulates the high bone mass phenotype of global Shn3 KO mice, including reduced osteoclastic bone catabolism in vivo, indicating that Shn3 expression in mesenchymal cells directly controls osteoblastic bone formation and indirectly regulates osteoclastic bone resorption."
07/01/2005 - "Taken together, these results indicate that the Ltbp-3-/- high bone mass phenotype was due to a defect in bone resorption. "
01/01/1990 - "The excellent therapeutic response confirmed by histological study of the residual masses, which show a fibrotic or necrotic appearance, allows the adoption of a strategy using imaging techniques: either there is persistence of a gland mass syndrome and resection needs to be carried our or retroperitoneal fibrosis is visualised and careful follow up would appear to be sufficient."
|1.||Angiotensin Receptor Antagonists
|5.||Neuraminidase deficiency with beta-galactosidase deficiency
|6.||Familial ectopia lentis
|8.||Islet Amyloid Polypeptide
|1.||Positive-Pressure Respiration (PEEP)
|3.||Drug Therapy (Chemotherapy)