|1.||Guh, Jih-Hwa: 1 article (05/2014)|
|2.||Lee, Yean-Jang: 1 article (05/2014)|
|3.||Kung, Fan-Lu: 1 article (05/2014)|
|4.||Hsu, Jui-Ling: 1 article (05/2014)|
|5.||Dong, Yu-Shun: 1 article (05/2014)|
|6.||Chen, Ching-Shih: 1 article (05/2014)|
|7.||Liu, Fan-Lun: 1 article (05/2014)|
|8.||Chen, Shu-Li: 1 article (01/2012)|
|9.||Chen, Guan-Yu: 1 article (01/2012)|
|10.||Lee, Chia-Lin: 1 article (01/2012)|
01/01/2012 - "In a structure-activity relationship (SAR) study, 3-methoxy-1,4-phenanthrenequinones, calanquinone A (6a), denbinobin (6b), 5-OAc-calanquinone A (7a) and 5-OAc-denbinobin (7b), have significantly promising cytotoxicity against various human cancer cell lines (IC(50) 0.08-1.66 µg/mL). "
|2.||Glioblastoma (Glioblastoma Multiforme)
05/05/2014 - "Calanquinone A, an herbal constituent, displayed anti-proliferative activity against glioblastoma cells, including A172, T98 and U87. "
05/05/2014 - "Furthermore, calanquinone A does not serve as a P-glycoprotein substrate, suggesting a potential for further development in anti-glioblastoma therapy. "
05/05/2014 - "In summary, the data suggest that calanquinone A displays anti-glioblastoma activity through a decrease of cellular glutathione levels that subsequently induces DNA damage stress and AMPK activation, leading to cell cycle arrest at S-phase and apoptotic cell death. "
05/05/2014 - "Calanquinone A induces anti-glioblastoma activity through glutathione-involved DNA damage and AMPK activation."
|3.||Glutathione (Reduced Glutathione)