|1.||Dyson, Paul J: 4 articles (01/2015 - 09/2008)|
|2.||Weiss, Andrea: 1 article (01/2015)|
|3.||Wong, Tse J: 1 article (01/2015)|
|4.||Griffioen, Arjan W: 1 article (01/2015)|
|5.||Scherrer, Edoardo: 1 article (01/2015)|
|6.||Nowak-Sliwinska, Patrycja: 1 article (01/2015)|
|7.||Bonvin, Débora: 1 article (01/2015)|
|8.||Berndsen, Robert H: 1 article (01/2015)|
|9.||Blunden, Bianca M: 1 article (12/2013)|
|10.||Lu, Hongxu: 1 article (12/2013)|
|1.||Ovarian Neoplasms (Ovarian Cancer)
12/09/2013 - "RAPTA-C conjugation, micellization, and subsequent cytotoxicity and cell uptake of these polymeric moieties was tested on ovarian cancer A2780, A2780cis, and Ovcar-3 cell lines. "
10/14/2010 - "The complexes showed a cytotoxic effect on human ovarian cancer cells and were markedly more active than their Ti or Ru monometallic analogues titanocene dichloride and RAPTA-C, respectively. "
09/01/2008 - "Consequently, RAPTA-C treatment results in a significant inhibition in the progression of cancer in an animal model, which emulates the human disease, and does so with remarkably low general toxicity; hence, RAPTA-C has potential for clinical application."
09/01/2008 - "RAPTA-C exhibits effective cell growth inhibition by triggering G(2)/M phase arrest and apoptosis in cancer cells. "
01/01/2015 - "Treatment with the chemotherapeutics, doxorubicin and RAPTA-C, as well as applying photodynamic therapy during this period of elevated oxygenation led to enhanced tumor growth inhibition. "
|4.||Breast Neoplasms (Breast Cancer)
05/01/2010 - "In vitro ruthenation of human breast cancer suppressor gene 1 (BRCA1) by the antimetastasis compound RAPTA-C and its analogue CarboRAPTA-C."
05/01/2010 - "The interaction of two ruthenium-arene-1,3,5-triaza-7-phosphaadamantane compounds ([Ru(eta(6)-p-cymene)Cl(2)(pta)] and [Ru(eta(6)-p-cymene)(C(6)H(6)O(4))(pta)], termed RAPTA-C (3) and carboRAPTA-C (4), resp.) with the DNA sequence of the human breast-cancer suppressor gene 1 (BRCA1) has been studied using a range of techniques that probe conformation, cross-linking, base specificity, restriction analysis, and in vitro inhibition of DNA polymerization. "
|4.||DNA (Deoxyribonucleic Acid)
|5.||Aminocaproic Acids (EAC)
|1.||Photochemotherapy (Photodynamic Therapy)