|1.||Portugal, José: 4 articles (10/2015 - 09/2008)|
|2.||Salas, José A: 3 articles (11/2012 - 09/2008)|
|3.||Méndez, Carmen: 3 articles (11/2012 - 09/2008)|
|4.||Vizcaíno, Carolina: 2 articles (10/2015 - 11/2012)|
|5.||Morís, Francisco: 2 articles (10/2015 - 11/2012)|
|6.||Núñez, Luz-Elena: 2 articles (10/2015 - 11/2012)|
|7.||Bataller, Marc: 2 articles (06/2010 - 09/2008)|
|8.||Rodríguez-Sánchez, Maria A: 1 article (10/2015)|
|9.||Scott, Daniel: 1 article (05/2013)|
|10.||Bae, Younsoo: 1 article (05/2013)|
06/01/2010 - "Cellular response and activation of apoptosis by mithramycin SK in p21(WAF1)-deficient HCT116 human colon carcinoma cells."
09/01/2008 - "Here, we show that treatment of HCT-116 (p53+/+) colon carcinoma cells with the novel antitumor antibiotic mithramycin SK (MSK) results in polyploidization and mitotic catastrophe, which occurs after a transient halt in G1 phase followed by the overtaking of the G2-M checkpoint when treated cells are incubated in a fresh drug-free medium. "
09/01/2008 - "Mithramycin SK modulates polyploidy and cell death in colon carcinoma cells."
10/01/2015 - "DIG-MSK (demycarosil-3D-β-D-digitoxosyl mithramycin SK; EC-8042), a novel analogue of mithramycin A, induced autophagy in HCT116 human colon carcinoma and, to a lesser extent, in A2780 human ovarian carcinoma cell lines, which was followed by apoptosis and/or necrotic cell death in a time-dependent way. "
11/01/2012 - "The effects of mithramycin SK (MSK) and demycarosyl-3D-β-D-digitoxosyl-mithramycin SK (DIG-MSK; EC-8042), two novel analogs of the antitumor antibiotic mithramycin A, on gene transcription were examined in human HCT116 colon carcinoma cells by quantitative real-time PCR of 89 genes mainly involved in cell cycle control. "
06/01/2010 - "HCT116 (p21(-/-)) human colon carcinoma cells treated with mithramycin SK (MSK), a novel analog of the antitumor antibiotic mithramycin A (MTA), were transiently arrested in G2/M, with some cells entering a faulty mitotic cycle without cytokinesis that resulted in G1-like cell arrest, which consisted of post-mitotic aneuploid G1 cells. "