|1.||Navakauskiene, Ruta: 4 articles (12/2012 - 11/2006)|
|2.||Magnusson, Karl-Eric: 3 articles (12/2012 - 11/2006)|
|3.||Borutinskaite, Veronika V: 2 articles (12/2012 - 12/2006)|
|4.||Savickiene, Jurate: 2 articles (01/2012 - 11/2006)|
|5.||Treigyte, Grazina: 2 articles (01/2012 - 11/2006)|
|6.||Borutinskaite, Veronika-Viktorija: 2 articles (01/2012 - 11/2006)|
|7.||Borutinskaitė, Veronika: 1 article (01/2015)|
|8.||Navakauskienė, Rūta: 1 article (01/2015)|
|9.||Jonusiene, Violeta: 1 article (01/2012)|
|10.||Bruzaite, Renata: 1 article (01/2012)|
01/01/2015 - "In this study we also examined how expression and activity of HDACs are affected after leukemia cell treatment with BML-210. "
11/07/2006 - "In this study, we examined the in vitro effects of BML-210 on the human leukemia cell lines (NB4, HL-60, THP-1, and K562). "
01/01/2015 - "Based on these and other findings from our group, we suggest that HDACIs, like BML-210, can be promising anticancer agents in promyelocytic leukemia treatment. "
01/01/2015 - "The Histone Deacetylase Inhibitor BML-210 Influences Gene and Protein Expression in Human Promyelocytic Leukemia NB4 Cells via Epigenetic Reprogramming."
11/07/2006 - "The novel histone deacetylase inhibitor BML-210 exerts growth inhibitory, proapoptotic and differentiation stimulating effects on the human leukemia cell lines."
12/01/2012 - "In conclusion, these results suggest that HDACI BML-210 can inhibit cell growth and induce apoptosis in cervical cancer cells, what correlates with down-regulation of HDAC class I and II and changes in the DAPC expression levels. "
12/01/2012 - "Histone deacetylase inhibitor BML-210 induces growth inhibition and apoptosis and regulates HDAC and DAPC complex expression levels in cervical cancer cells."
12/01/2006 - "Retinoic acid and histone deacetylase inhibitor BML-210 inhibit proliferation of human cervical cancer HeLa cells."
01/01/2012 - "Pretreatment for 24 or 48 h with zebularine before the treatment with different doses of RA alone or RA with histone deacetylase inhibitors, phenyl butyrate, and BML-210, resulted in significant acceleration and enhancement of differentiation and cell cycle arrest at G0/1. Zebularine alone or in sequential combination with RA decreased expression of DNMT1, caused fast and time-dependent expression of pan-cadherin and partial demethylation of E-cadherin but not tumor suppressor p15. "
|1.||Histone Deacetylase Inhibitors
|3.||Tretinoin (Retinoic Acid)
|5.||1,2- diarachidonoyl- glycero- 3- phosphocholine