|1.||Brusilow, William S: 1 article (01/2010)|
|2.||Chiara, Federica: 1 article (01/2010)|
|3.||Bernardi, Paolo: 1 article (01/2010)|
|4.||Rasola, Andrea: 1 article (01/2010)|
|5.||Pantic, Boris: 1 article (01/2010)|
|6.||Sciacovelli, Marco: 1 article (01/2010)|
|7.||Brabant, Magali: 1 article (01/2010)|
|8.||Racoeur, Cindy: 1 article (01/2010)|
|9.||Péchoux, Christine: 1 article (01/2010)|
|10.||Lassalle, Myriam: 1 article (01/2010)|
01/12/2010 - "All these tumor cells displayed marked resistance to death caused by apoptotic stimuli like arachidonic acid and the BH3 mimetic EM20-25, which cause cell death through the mitochondrial permeability transition pore (PTP). "
01/01/2010 - "The truncated (t-) Bid protein, synthetic BH3 peptides from Bim and Bak, and the small molecule ABT-737 induced a tumor-specific and OMP-restricted mitochondrio-toxicity, while compounds like HA-14.1, YC-137, Chelerythrine, Gossypol, TW-37 or EM20-25 did not. "
09/01/2008 - "In a direct comparison of peripheral blood mononuclear cells from healthy volunteers with primary CLL, the two agents combined showed a therapeutic index of 19-fold; furthermore, the combination of VSV and EM20-25 increased apoptotic cell death in Karpas-422 and Granta-519 B-lymphoma cell lines (P < 0.005) via the intrinsic mitochondrial pathway. "
|1.||BH3 Interacting Domain Death Agonist Protein
|2.||Arachidonic Acid (Vitamin F)
|6.||mitochondrial permeability transition pore