|1.||Zhang, Yuelin: 1 article (02/2013)|
|2.||Tse, Hung-Fat: 1 article (02/2013)|
|3.||Chan, Yau-Chi: 1 article (02/2013)|
|4.||Liu, Yuan: 1 article (02/2013)|
|5.||Li, Ronald A: 1 article (02/2013)|
|6.||Mei-Chu Yip, Pandora: 1 article (02/2013)|
|7.||Liao, Song-Yan: 1 article (02/2013)|
|8.||Shin, Ki Soon: 1 article (02/2008)|
|9.||Yoo, Soon Ji: 1 article (02/2008)|
|10.||Cho, Sang-Hee: 1 article (02/2008)|
04/15/2005 - "Short QT syndrome or Andersen syndrome: Yin and Yang of Kir2.1 channel dysfunction."
07/01/2005 - "Loss of function in the slow component of the delayed rectifier potassium current (I(Ks)) channels (KCNQ1, KCNE1), the rapid component of the potassium current (I(Kr)) channels (KCNH2, KCNE2) and the inward rectifier potassium current (I(Kl), Kir2.1) channel (KCNJ2) is linked to the LQTSs (type 1, 2, 5, 6, and 7 (Andersen syndrome)) and the JLNSs (type 1 and 2). "
02/29/2008 - "However, Kir2.1 channel-expressing cells were dramatically increased in the hippocampus by kainic acid-induced seizure and the cells were glial fibrillary acidic protein (GFAP)-positive, which confirms that astrocytes in the hippocampus express Kir2.1 channels under pathological conditions. "
|2.||Kainic Acid (Kainate)
|3.||Glial Fibrillary Acidic Protein