|1.||Ye, Keqiang: 4 articles (03/2015 - 02/2010)|
|2.||Liu, Xia: 3 articles (03/2015 - 02/2010)|
|3.||Choi, Yung Hyun: 3 articles (02/2015 - 07/2012)|
|4.||Kim, Gi-Young: 2 articles (03/2013 - 07/2012)|
|5.||Park, Hye Young: 2 articles (03/2013 - 07/2012)|
|6.||Yoo, Young Hyun: 2 articles (03/2013 - 07/2012)|
|7.||Kim, Nam Deuk: 2 articles (03/2013 - 07/2012)|
|8.||Churchill, M J: 1 article (04/2015)|
|9.||Sconce, M D: 1 article (04/2015)|
|10.||Moore, C: 1 article (04/2015)|
|1.||Ganglion Cysts (Ganglion)
|2.||Rett Syndrome (Rett's Disorder)
04/01/2014 - "The results from in vitro studies showed that 7,8-dihydroxyflavone (7,8-DHF), a TrkB receptor agonist, upregulates the levels of biogenesis activator PGC-1α, and CREB phosphorylation in neuroblastoma cells suggesting that BDNF-TrkB signaling is pivotal for engaging signals related to synaptic plasticity and energy metabolism. "
|4.||Brain Injuries (Brain Injury)
01/01/2014 - "The present study investigated the effects and underlying mechanisms of TrkB activation by the TrkB agonist 7,8-dihydroxyflavone (7,8-DHF) on traumatic brain injury (TBI). "
01/01/2014 - "Post-injury treatment with 7,8-dihydroxyflavone, a TrkB receptor agonist, protects against experimental traumatic brain injury via PI3K/Akt signaling."
|5.||Parkinson Disease (Parkinson's Disease)
04/02/2015 - "Intervention with 7,8-dihydroxyflavone blocks further striatal terminal loss and restores motor deficits in a progressive mouse model of Parkinson's disease."
01/01/2015 - "It is reported that 7,8-dihydroxyflavone (DHF), a TrkB agonist, has beneficial effects on neuronal excitotoxicity, stroke, and Parkinson disease in animal models by enhancing axon regeneration, muscle reinnervation and neuromuscular transmission. "
07/25/2011 - "7,8-Dihydroxyflavone (7,8-DHF), a member of the flavonoid family, is a selective tyrosine kinase receptor B (TrkB) agonist that has neurotrophic effects in various neurological diseases such as stroke and Parkinson's disease. "
02/09/2010 - "Administration of 7,8-dihydroxyflavone to mice activated TrkB in the brain, inhibited kainic acid-induced toxicity, decreased infarct volumes in stroke in a TrkB-dependent manner, and was neuroprotective in an animal model of Parkinson disease. "
|3.||Brain-Derived Neurotrophic Factor (BDNF)
|5.||Proteins (Proteins, Gene)
|6.||Cyclin-Dependent Kinase Inhibitor p27
|8.||RNA (Ribonucleic Acid)
|9.||Protein-Tyrosine Kinases (Tyrosine Kinase)