|1.||Adler, Kenneth B: 3 articles (09/2010 - 02/2004)|
|2.||Adler, K B: 2 articles (07/2014 - 01/2007)|
|3.||Park, Joungjoa: 2 articles (06/2006 - 02/2004)|
|4.||Chen, Ching-Hsien: 1 article (06/2015)|
|5.||Fong, Lon Wolf R: 1 article (06/2015)|
|6.||Yuan, Yuan: 1 article (06/2015)|
|7.||Cheng, Chun-Ting: 1 article (06/2015)|
|8.||Ann, David K: 1 article (06/2015)|
|9.||Wu, Reen: 1 article (06/2015)|
|10.||Arif, Muhammad: 1 article (06/2015)|
|1.||Asthma (Bronchial Asthma)
06/01/2006 - "J Biol Chem 2001;276:40982-40990; Singer M, Martin LD, Vargaftig BB, Park J, Gruber AD, Li Y, Adler KB. A MARCKS-related peptide blocks mucus hypersecretion in a mouse model of asthma. "
02/01/2004 - "A MARCKS-related peptide blocks mucus hypersecretion in a mouse model of asthma."
02/01/2004 - "Intratracheal administration of this MARCKS-related peptide could therapeutically reduce mucus secretion in the airways of human patients with asthma, chronic bronchitis and cystic fibrosis."
01/01/2007 - "Inhibition of mucin secretion with MARCKS-related peptide improves airway obstruction in a mouse model of asthma."
|3.||Neoplasm Metastasis (Metastasis)
09/01/2010 - "Collectively, these findings support the concept that even in airway epithelia remodeled with goblet cell metaplasia and in a state of mucin hypersecretion, exogenous attenuation of function of MARCKS protein via the MANS peptide decreases airway mucin secretion, inflammation, and hyperreactivity."
09/01/2010 - "In a mouse model of neutrophil elastase-induced bronchitis that exhibits goblet cell metaplasia and inflammation, we investigated the effects of intratracheal instillation of the MANS peptide, a peptide identical to the NH(2) terminus of the myristoylated alanine-rich C kinase substrate (MARCKS) on mucin protein airway secretion, inflammation, and airway reactivity. "
|5.||Lung Neoplasms (Lung Cancer)
|2.||Leukocyte Elastase (Neutrophil Elastase)
|4.||myristoylated alanine-rich C kinase substrate (MARCKS protein)