|1.||Rioli, Vanessa: 4 articles (12/2007 - 03/2003)|
|2.||Ferro, Emer S: 3 articles (06/2014 - 03/2003)|
|3.||Heimann, Andrea S: 3 articles (06/2014 - 03/2003)|
|4.||Dale, Camila S: 2 articles (06/2014 - 12/2007)|
|5.||Devi, Lakshmi A: 2 articles (06/2014 - 12/2007)|
|6.||Lutz, Beat: 2 articles (08/2013 - 05/2010)|
|7.||Luckman, Simon M: 2 articles (08/2013 - 05/2010)|
|8.||Dodd, Garron T: 2 articles (08/2013 - 05/2010)|
|9.||Giorgi, Renata: 2 articles (12/2007 - 03/2005)|
|10.||Hyslop, Stephen: 2 articles (03/2005 - 03/2003)|
08/01/2013 - "Thus, hemopressin modulates the function of key feeding-related brain nuclei of the mediobasal hypothalamus, and descending pain pathways of the PAG and DR, and not higher limbic structures. "
12/18/2007 - "Finally, we examine the activity of hemopressin in vivo using different models of pain and find that it exhibits antinociceptive effects when administered by either intrathecal, intraplantar, or oral routes, underscoring hemopressin's therapeutic potential. "
03/01/2005 - "In this work, we examined the influence of hemopressin on inflammatory pain. "
11/01/2012 - "The current study evaluated the effects of hemopressin (HP) on pain modulation by endokinin A/B (EKA/B) and endokinin C/D (EKC/D) at the supraspinal level in mice. "
06/01/2014 - "Hemopressin (Hp) is a nonapeptide that selectively binds CB1 cannabinoid receptors (CB1 receptors) and exerts antinociceptive action in inflammatory pain models. "
|2.||Hypotension (Low Blood Pressure)
03/01/2005 - "Hemopressin causes hypotension in anesthetized rats and is metabolized in vivo and in vitro by endopeptidase 24.15 (EP24.15), neurolysin (EP24.16), and angiotensin-converting enzyme (ACE). "
03/07/2003 - "The hemoglobin alpha-chain fragment PVNFKFLSH, which we have named hemopressin, produced dose-dependent hypotension in anesthetized rats, starting at 0.001 microg/kg. The hypotensive effect of the peptide was potentiated by enalapril only at the lowest peptide dose. "
03/01/2005 - "Antinociceptive action of hemopressin in experimental hyperalgesia."
03/01/2005 - "Hemopressin administered systemically also reverted the hyperalgesia induced by carrageenin. "
03/01/2005 - "Hemopressin reverted the hyperalgesia induced by either carrageenin or bradykinin when injected concomitantly or 2.5 h after the phlogistic agents. "
03/01/2005 - "Hemopressin also exerts an antinociceptive action in experimental inflammatory hyperalgesia induced by carrageenin or bradykinin via a mechanism that is independent of opioids. "
04/01/2014 - "Considering diversity of endogenous neurolysin substrates (neurotensin, bradykinin, angiotensins I/II, substance P, hemopressin, dynorphin A(1-8), metorphamide, somatostatin) and the well-documented role of these peptidergic systems in pathogenesis of stroke, resistance to ischemic injury and/or post-stroke brain recovery, our findings suggest that neurolysin may play a role in processes modulating the brain's response to stroke and its recovery after stroke."
|5.||Neuralgia (Stump Neuralgia)
|1.||2'- deoxythymidylyl- (3'- 5')- 2'- deoxyadenosine (d(AT))
|3.||CB1 Cannabinoid Receptor (CB1 Receptor)
|8.||CB2 Cannabinoid Receptor
|9.||Cannabinoid Receptors (Cannabinoid Receptor)