|1.||Kim, Hyun J: 1 article (04/2014)|
|2.||Kim, Young J: 1 article (04/2014)|
|3.||Bae, Yong C: 1 article (04/2014)|
|4.||Ahn, Dong K: 1 article (04/2014)|
|5.||Kim, Min J: 1 article (04/2014)|
|6.||Lee, Sang Y: 1 article (04/2014)|
|7.||Nam, Soon H: 1 article (04/2014)|
|8.||Yang, Kui Y: 1 article (04/2014)|
|9.||Jaffal, S M: 1 article (09/2009)|
|10.||El-Hashim, A Z: 1 article (09/2009)|
05/25/2005 - "We show here that acute pain-related behavior evoked by elevated ionic strength is abolished in TRP vanilloid subtype 1 (TRPV1)-null mice and inhibited by iodoresiniferatoxin, a potent TRPV1 antagonist. "
04/01/2006 - "The purpose of this study is to investigate the participation of spinal TRPV1 in formalin-evoked pain with iodo-resiniferatoxin (I-RTX), a potent TRPV1-selective antagonist. "
04/01/2006 - "Participation of the spinal TRPV1 receptors in formalin-evoked pain transduction: a study using a selective TRPV1 antagonist, iodo-resiniferatoxin."
04/18/2003 - "These effects were blocked by co-administration of the VR1 antagonist iodo-resiniferatoxin (10 nm for HEK cells and 1 microg/50 micro;l for pain behavior). "
01/01/2007 - "The VR1 antagonists capsazepine and iodo-resiniferatoxin (IRTX) did not inhibit cough in either model. "
09/01/2009 - "Pretreatment with the TrkA receptor antagonist, K252a, or the TRPV1 antagonist, iodoresiniferatoxin, significantly inhibited the NGF-enhanced cough and airway obstruction. "
|3.||Myocardial Ischemia (Ischemic Heart Diseases)
|5.||Airway Obstruction (Choking)
|2.||Nerve Growth Factor (NGF)
|6.||K 252 (K252a)