|1.||Sorrentino, Rosa: 6 articles (01/2012 - 03/2003)|
|2.||Ziegler, Andreas: 6 articles (08/2008 - 01/2004)|
|3.||Uchanska-Ziegler, Barbara: 6 articles (02/2008 - 01/2004)|
|4.||Fiorillo, Maria Teresa: 5 articles (01/2012 - 05/2003)|
|5.||Mathieu, Alessandro: 4 articles (01/2012 - 03/2003)|
|6.||Böckmann, Rainer A: 4 articles (01/2012 - 07/2004)|
|7.||Alexiev, Ulrike: 4 articles (08/2008 - 07/2004)|
|8.||Narzi, Daniele: 3 articles (01/2012 - 02/2008)|
|9.||Cauli, Alberto: 3 articles (01/2012 - 05/2003)|
|10.||Misselwitz, Rolf: 3 articles (08/2008 - 03/2005)|
08/01/2007 - "The HLA-B*2709 subtype in a woman with early ankylosing spondylitis."
01/01/2012 - "The single amino acid replacement Asp116His distinguishes the two subtypes HLA-B*2705 and HLA-B*2709 which are, respectively, associated and non-associated with Ankylosing Spondylitis, an autoimmune chronic inflammatory disease. "
01/28/2005 - "It could also be involved in the differential association of the human major histocompatibility subtypes HLA-B(*)2705 and HLA-B(*)2709 with ankylosing spondylitis. "
05/01/2003 - "To compare haplotype distribution in HLA-B27-positive patients with ankylosing spondylitis (AS) and healthy control subjects possessing either AS-associated HLA-B27 alleles or the non-AS-associated HLA-B*2709 allele. "
08/01/1998 - "The naturally occurring polymorphism Asp116-->His116, differentiating the ankylosing spondylitis-associated HLA-B*2705 from the non-associated HLA-B*2709 subtype, influences peptide-specific CD8 T cell recognition."
|2.||Autoimmune Diseases (Autoimmune Disease)
07/02/2004 - "Here we show that the nanosecond dynamics of this peptide presented by two human MHC class I subtypes (HLA-B*2705 and HLA-B*2709) with differential autoimmune disease association varies dramatically, despite virtually identical crystal structures. "
08/22/2008 - "A single amino acid exchange between the major histocompatibility complex molecules HLA-B(*)2705 and HLA-B(*)2709 (Asp-116/His) is responsible for the emergence of distinct HLA-B27-restricted T cell repertoires in individuals harboring either of these two subtypes and could correlate with their differential association with the autoimmune disease ankylosing spondylitis. "
10/15/2007 - "A single natural amino-acid substitution distinguishes the HLA-B*2705 subtype that is associated with the autoimmune disease ankylosing spondylitis from the non-disease-associated HLA-B*2709 subtype. "
03/11/2005 - "Residues forming this pocket exhibit considerable polymorphism, and a single difference (Asp116 in HLA-B*2705 and His116 in HLA-B*2709 heavy chains) confers differential association of these two HLA-B27 subtypes to the autoimmune disease ankylosing spondylitis. "
01/19/2004 - "The products of the human leukocyte antigen subtypes HLA-B*2705 and HLA-B*2709 differ only in residue 116 (Asp vs. His) within the peptide binding groove but are differentially associated with the autoimmune disease ankylosing spondylitis (AS); HLA-B*2705 occurs in AS-patients, whereas HLA-B*2709 does not. "
11/01/2008 - "HLA-B*2709 and lack of susceptibility to sacroiliitis: further support from the clinic."
11/01/2008 - "We describe the unique case of a HLA-B*2709 positive subject who underwent IFN-alpha treatment for essential thrombocythemia and developed arthritis of the proximal interphalangeal joints of the hands but not sacroiliitis. "
|5.||Spondylarthritis (Spinal Arthritis)
|1.||HLA-B 2705 antigen
|2.||HLA-B27 Antigen (HLA B27 Antigen)
|3.||HLA-B Antigens (HLA-B)
|4.||Proteins (Proteins, Gene)