|1.||Ziegler, Andreas: 6 articles (08/2008 - 01/2004)|
|2.||Uchanska-Ziegler, Barbara: 6 articles (02/2008 - 01/2004)|
|3.||Fiorillo, Maria Teresa: 4 articles (01/2012 - 01/2004)|
|4.||Böckmann, Rainer A: 4 articles (01/2012 - 07/2004)|
|5.||Sorrentino, Rosa: 4 articles (01/2012 - 01/2004)|
|6.||Alexiev, Ulrike: 4 articles (08/2008 - 07/2004)|
|7.||Narzi, Daniele: 3 articles (01/2012 - 02/2008)|
|8.||Osterhaus, A D M E: 3 articles (01/2011 - 01/2002)|
|9.||Rimmelzwaan, G F: 3 articles (01/2011 - 01/2002)|
|10.||Misselwitz, Rolf: 3 articles (08/2008 - 03/2005)|
12/01/2005 - "In contrast, KIR gene repertoire had no effect on pretreatment viral load while selected HLA alleles (eg HLA-B*5701, HLA-B*2705) demonstrated significant protective effects on viremia. "
06/15/2009 - "In a similar fashion, 50% of Mamu-B*08-positive Indian rhesus macaques control SIVmac239 replication and become elite controllers with chronic-phase viremia <1000 viral RNA copies/ml. Interestingly, Mamu-B*08-restricted SIV-derived epitopes appeared to match the peptide binding profile for HLA-B*2705 in humans. "
02/01/2010 - "Association of ARTS1 gene polymorphisms with ankylosing spondylitis in the Hungarian population: the rs27044 variant is associated with HLA-B*2705 subtype in Hungarian patients with ankylosing spondylitis."
12/01/2003 - "The frequency of the HLA-B*2705 allele among the B27 positive uSpA patients of this series was closely similar to that reported for patients with ankylosing spondylitis (AS). "
08/09/2002 - "In contrast to HLA-B*2705, B*2709 is weakly or not associated to ankylosing spondylitis. "
08/09/2002 - "Differential association of HLA-B*2705 and B*2709 to ankylosing spondylitis correlates with limited peptide subsets but not with altered cell surface stability."
09/01/1999 - "To investigate the rules governing peptide binding to HLA-B*2705, and to B*2704 and B*2706, which are 2 subtypes differentially associated with ankylosing spondylitis. "
01/01/2011 - "In this study, we examined Ag-specific CD8(+) TCR repertoires longitudinally in a cohort of HLA-B*2705(+) long-term nonprogressors with chronic HIV-1 infection using a combination of molecular clonotype analysis and polychromatic flow cytometry. "
01/01/2011 - "In contrast, HLA-B*2705 transgenic mice primed for a secondary CTL response by infection with a heterosubtypic influenza A virus, did not display this difference in virulence and the outcome of infection with the 384R virus was somewhat reduced. "
07/01/2001 - "A protocol was developed for efficient large-scale infection of lymphoblastoid cell transfectants expressing HLA-B*2705. "
01/01/2011 - "Infection of naïve C57Bl/6 and HLA-B*2705 mice with influenza virus containing the NP(383-391) epitope (384R) caused more weight loss compared to infection with the virus without the epitope (384G). "
|4.||Autoimmune Diseases (Autoimmune Disease)
07/02/2004 - "Here we show that the nanosecond dynamics of this peptide presented by two human MHC class I subtypes (HLA-B*2705 and HLA-B*2709) with differential autoimmune disease association varies dramatically, despite virtually identical crystal structures. "
08/22/2008 - "A single amino acid exchange between the major histocompatibility complex molecules HLA-B(*)2705 and HLA-B(*)2709 (Asp-116/His) is responsible for the emergence of distinct HLA-B27-restricted T cell repertoires in individuals harboring either of these two subtypes and could correlate with their differential association with the autoimmune disease ankylosing spondylitis. "
10/15/2007 - "A single natural amino-acid substitution distinguishes the HLA-B*2705 subtype that is associated with the autoimmune disease ankylosing spondylitis from the non-disease-associated HLA-B*2709 subtype. "
09/01/1998 - "High-affinity ligands of non-peptidic nature, binding to the class I major histocompatibility complex protein HLA B*2705 whose expression is strongly linked to the pathogenesis of the autoimmune disease ankylosing spondylitis, should give way to a selective immunotherapy by blocking or antagonising the interaction with autoreactive T cell clones. "
03/11/2005 - "Residues forming this pocket exhibit considerable polymorphism, and a single difference (Asp116 in HLA-B*2705 and His116 in HLA-B*2709 heavy chains) confers differential association of these two HLA-B27 subtypes to the autoimmune disease ankylosing spondylitis. "
|5.||Human Influenza (Influenza)
01/01/2011 - "Redundancy of the influenza A virus-specific cytotoxic T lymphocyte response in HLA-B*2705 transgenic mice limits the impact of a mutation in the immunodominant NP(383-391) epitope on influenza pathogenesis."
05/01/2004 - "A mutation in the HLA-B*2705-restricted NP383-391 epitope affects the human influenza A virus-specific cytotoxic T-lymphocyte response in vitro."
01/01/2011 - "During the 1993-1994 flu season, influenza A/H3N2 viruses emerged with an amino acid substitution (R384G) at the anchor residue of the HLA-B*2705 restricted NP(383-391) epitope located in the nucleoprotein (NP). "
02/01/2005 - "We have solved the crystal structures of three HLA-B*2705-peptide complexes with the immunodominant viral peptides: EBV EBNA3C 258-266 (RRIYDLIEL), influenza (flu) nucleoprotein NP383-391 (SRYWAIRTR), and HIV gag 264-273 (KRWIILGLNK). "
01/01/2002 - "Furthermore, N-acetyl-L-cysteine was found to enhance a specific activity of two influenza specific CD8+ cytotoxic T-lymphocyte clones directed towards HLA-A*0201 and HLA-B*2705 restricted epitopes. "
|1.||HLA-B Antigens (HLA-B)
|2.||HLA-B27 Antigen (HLA B27 Antigen)
|3.||HLA-B 2709 antigen
|8.||Collagen Type XI
|10.||Proteins (Proteins, Gene)