|1.||Zhao, Jing: 2 articles (07/2013 - 01/2010)|
|2.||Zhao, Yong: 2 articles (07/2013 - 01/2010)|
|3.||Liu, Ke Jian: 2 articles (03/2003 - 05/2002)|
|4.||Liu, Shimin: 2 articles (03/2003 - 05/2002)|
|5.||Miyake, Minoru: 2 articles (03/2003 - 05/2002)|
|6.||Liu, Miao: 2 articles (03/2003 - 05/2002)|
|7.||Chen, Yanlin: 1 article (07/2013)|
|8.||Lei, Shipeng: 1 article (07/2013)|
|9.||Wu, Xuemei: 1 article (07/2013)|
|10.||Yu, Shanshan: 1 article (07/2013)|
01/13/2010 - "Neurological evaluation and Nissl-staining of the 4-HBA group were improved significantly compared to the untreated ischemia group. "
01/13/2010 - "Under the treatment of 50 mg/kg 4-HBA, total (100.76+/-2.90 mm(3)), cortical (64.91+/-1.46 mm(3)), and sub-cortical (38.77+/-2.78 mm(3)) infarct volumes were significantly decreased in comparison to ischemia-reperfusion values. "
11/21/2007 - "Treatment with 4-HBA did not affect NR1 expression levels, down-regulated 8-OHdG immunoreactivity, and increased GABA-T expression levels after global ischemia, suggesting that 4-HBA inhibited NR1 stimulation. "
03/15/2003 - "After microdialysis probe delivery of 4-HBA, (*)OH production was significantly increased in the striatal core during both ischemia and reperfusion. "
|2.||Brain Ischemia (Cerebral Ischemia)
07/01/2013 - "Furthermore, 4-HBA reduced the cerebral infarct size and improved the behavioral parameters after cerebral ischemia. "
01/13/2010 - "These results suggested that 4-HBA ameliorated ischemic injury induced by transient focal cerebral ischemia in rats, and this neuroprotective effect may be partly related to attenuate apoptosis pathway."
05/01/2002 - "Because of interrupted blood flow during cerebral ischemia and reperfusion, delivery of 4-HBA through the microdialysis probe is a preferred method to systemic administration such as intravenous infusion. "
06/01/2009 - "Only the stroke protective 4-HBA, but neither 3-HBA nor 2-HBA, was capable of significantly inducing PDI in intact mouse brains. "
06/01/2009 - "Stroke protection was fully prevented by bacitracin (500 mg/kg), a known inhibitor of PDI, which, without affecting basal brain PDI levels, altered the ability of 4-HBA to induce significantly PDI in intact brains. "
06/01/2009 - "Taken as a whole, our data indicate that stroke protection induced by 4-HBA involves PDI as a key player, making this protein a valuable target to control brain injury disorders. "
|4.||Nervous System Diseases (Neurological Disorders)
|1.||Staphylococcal Protein A (A, Protein)
|2.||gamma-Aminobutyric Acid (GABA)