|1.||Saenko, A S: 2 articles (07/2008 - 05/2005)|
|2.||Semina, O V: 2 articles (07/2008 - 05/2005)|
|3.||Semenets, T N: 2 articles (07/2008 - 05/2005)|
|4.||Deigin, V I: 2 articles (07/2008 - 05/2002)|
|5.||Semin, D Yu: 1 article (07/2008)|
|6.||Iljina, T P: 1 article (07/2008)|
|7.||Zamulaeva, I A: 1 article (07/2008)|
|8.||Selivanova, E I: 1 article (07/2008)|
|9.||Maliutina, Ya V: 1 article (07/2008)|
|10.||Maliutina, Ia V: 1 article (05/2005)|
07/01/2008 - "We hypothesized that dipeptide gamma-d-Glu-d-Trp suppressed mobilization/migration of CD34+ hemopoietic precursor cells from the bone marrow to the peripheral blood of tumor-bearing mice."
07/01/2008 - "In tumor-bearing mice treated with gamma-d-Glu-d-Trp, the percent of CD34+ hemopoietic precursors in the peripheral blood considerably decreased throughout the observation period; the content of CD34+ hemopoietic precursors in the tumor tissue was 2-3-fold below the control against the background of increased content of CD34+ cells in the bone marrow. "
05/01/2005 - "On the contrary, Thymodepressin-treated tumor cells became even more heat-sensitive. "
05/01/2005 - "Was studied the influence of the Thymodepressin (dipeptide D-iEW--a new Russian immuno- and haemodepressant), on the hyperthermic sensitivity of haemopoietic precursors (CFU-S) and tumor model cells (EL-4 and Rauscher leukaemia). "
07/01/2008 - "Dipeptide gamma-d-Glu-d-Trp (thymodepressin) inhibits migration of CD34+ cells from the bone marrow into peripheral blood during tumor growth."
|2.||Atopic Dermatitis (Atopic Eczema)
05/01/2002 - "These findings suggest that disorders in keratinocyte proliferation are an important component in the pathogenesis of atopic dermatitis and confirm high efficiency of thymodepressin in the treatment of this condition."
05/01/2002 - "Proliferative processes in the epidermis of patients with atopic dermatitis treated with thymodepressin."
05/01/2002 - "Proliferative activity of the epidermis in skin biopsy specimens from patients with atopic dermatitis before and during therapy with thymodepressin (immunosuppressant) was studied by immunohistochemical method (by expression of Ki-67 antigen). "
|2.||Ki-67 Antigen (Ki 67 Antigen)