|1.||Suzuki, K: 3 articles (10/2011 - 04/2000)|
|2.||Goll, Darrel E: 3 articles (04/2007 - 05/2004)|
|3.||Thompson, Valery F: 3 articles (04/2007 - 05/2004)|
|4.||Koohmaraie, M: 3 articles (05/2004 - 06/2001)|
|5.||Shearer, T R: 3 articles (08/2000 - 03/2000)|
|6.||Fukiage, C: 3 articles (08/2000 - 03/2000)|
|7.||Azuma, M: 3 articles (08/2000 - 03/2000)|
|8.||Pomponio, Luigi: 2 articles (05/2012 - 11/2008)|
|9.||Ertbjerg, Per: 2 articles (05/2012 - 11/2008)|
|10.||Shimizu, K: 2 articles (10/2011 - 11/2004)|
02/01/1995 - "Studies on the autolysis of m-calpain from the skeletal muscle of the amphibian Rana ridibunda."
07/01/2011 - "We propose a model for m-calpain activation that does not involve either autolysis or subunit dissociation."
07/01/2011 - "m-Calpain activation in vitro does not require autolysis or subunit dissociation."
11/01/2008 - "longissimus dorsi from three pigs collected at different times during storage further confirmed post-mortem autolysis of m-calpain. "
11/01/2008 - "The results indicated post-mortem autolysis of m-calpain as two m-calpain bands were observed on the zymogram gel. "
12/06/2011 - "Thus, a strategy to selectively inhibit m-calpain may be useful for the therapeutic treatment of patients with atherosclerosis."
12/06/2011 - "m-Calpain induction in vascular endothelial cells on human and mouse atheromas and its roles in VE-cadherin disorganization and atherosclerosis."
08/17/2011 - "M-calpain may be involved in the preconditioning response to ischemia by upregulating endogenous pro-survival mechanisms in neurons."
04/01/1998 - "In the case of m-calpain, the only observed effect of ischemia was its redistribution and, as a consequence, the elevation of activity in particulate fraction. "
10/01/1997 - "m-Calpain was enriched in the nucleus-myofibril fraction but was not further translocated on ischemia-reperfusion. "
10/15/2015 - "Mitochondrial m-calpain opens the mitochondrial permeability transition pore in ischemia-reperfusion."
07/01/2010 - "Knockdown of micro-calpain, but not m-calpain, prevented calpain activity 72 h after 6-min transient forebrain ischemia, increased long-term survival and protected hippocampal electrophysiological function. "
10/01/2004 - "This finding was corroborated by immunohistochemical studies that showed strong cytoplasmic staining in the colon tumors with m-calpain antibody. "
02/29/2008 - "Ubiquitously expressed micro- and m-calpain are cysteine proteases with broad functions in cell spreading, migration, proliferation, apoptosis, and in tumor invasion. "
10/01/2004 - "Of 50 cases evaluated, we presented in this report six cases for m-calpain, calpastatin and HMWCaMBP protein expression by Western blot analyses was done in both normal and invasive tumor components of human samples. "
06/01/2000 - "The results presented clearly show that calpain-3 gene expression is considerably reduced in experimental cancer cachexia, while there is a reciprocal change in the expression of the ubiquitin-dependent proteolytic system and in the ubiquitous m-calpain. "
03/01/2011 - "There were no differences in mRNA levels for μ- and m-calpain, calpastatin, atrogin-1, or MuRF1 between control and cancer patients. "
|5.||Brain Injuries (Brain Injury)
02/01/1998 - "Subcellular localization and duration of mu-calpain and m-calpain activity after traumatic brain injury in the rat: a casein zymography study."
01/01/2000 - "Casein zymogram assessment of mu-calpain and m-calpain activity after traumatic brain injury in the rat in vivo."
05/08/1998 - "pH dependency of mu-calpain and m-calpain activity assayed by casein zymography following traumatic brain injury in the rat."
10/01/1998 - "One dimensional immunoblot of BDPs obtained from in vitro cleavage of enriched neurofilaments (NF) by purified micro-calpain, m-calpain, cathepsin, B, cathepsin D, and CPP32 (caspase-3) were compared to in vivo samples from rats following traumatic brain injury (TBI). "
02/01/1998 - "Casein zymographic assays were performed to identify changes in mu-calpain and m-calpain activity in naive, sham-injured, and injured rat cortex at 15 minutes, 3 hours, 6 hours, and 24 hours after unilateral cortical impact brain injury. "
|5.||Phosphatidic Acids (Phosphatidic Acid)
|6.||Choline (Choline Chloride)