|1.||Boland, Wilhelm: 1 article (04/2004)|
|2.||Maffei, Massimo: 1 article (04/2004)|
|3.||Bossi, Simone: 1 article (04/2004)|
|4.||Mithöfer, Axel: 1 article (04/2004)|
|5.||Spiteller, Dieter: 1 article (04/2004)|
|6.||Schmelz, Eric A: 1 article (09/2003)|
|7.||Engelberth, Juergen: 1 article (09/2003)|
|8.||Alborn, Hans T: 1 article (09/2003)|
|9.||Tumlinson, James H: 1 article (09/2003)|
|10.||Schmelz, E A: 1 article (12/2001)|
|1.||Wounds and Injuries (Trauma)
09/01/2003 - "As predicted, low N plants exhibited greater sustained increases in wound- and volicitin-induced JA levels compared with medium N plants. "
12/01/2001 - "Volicitin is hypothesized to trigger release of induced volatiles, at least in part, by modulating levels of the wound hormone, jasmonic acid (JA). "
|2.||Bites and Stings (Sting)
04/01/2004 - "The results showed that the early events upon herbivore attack were: a) a strong V(m) depolarization at the bite zone and an isotropic wave of V(m) depolarization spreading throughout the entire attacked leaf; b) a V(m) depolarization observed for the regurgitant but not with volicitin [N-(17-hydroxy-linolenoyl)-Gln] alone; c) an enhanced influx of Ca(2+) at the very edge of the bite, which is halved, if the Ca(2+) channel blocker Verapamil is used. "
|3.||jasmonic acid (jasmonate)