|1.||Wang, Yang: 3 articles (11/2007 - 02/2004)|
|2.||Lou, Kaiyan: 3 articles (11/2007 - 02/2004)|
|3.||Hua, Duy H: 3 articles (11/2007 - 02/2004)|
|4.||Perchellet, Elisabeth M: 3 articles (11/2007 - 02/2004)|
|5.||Perchellet, Jean-Pierre H: 2 articles (11/2007 - 10/2005)|
|6.||Zhao, Huiping: 1 article (11/2007)|
|7.||Battina, Srinivas K: 1 article (11/2007)|
|8.||Ward, Mary M: 1 article (10/2005)|
|9.||Weber, Rebeka L: 1 article (02/2004)|
|10.||Perchellet, Jean-Pierre: 1 article (02/2004)|
02/01/2004 - "Synthetic analogs of 1,4-anthraquinone (AQ code number), a compound that mimics the antiproliferative effects of daunorubicin (daunomycin) in the nanomolar range in vitro but has the advantage of blocking nucleoside transport and retaining its efficacy in multidrug-resistant tumor cells, were tested for their ability to induce apoptosis in the HL-60 cell system. "
10/01/2005 - "Since synthetic analogs of 1,4-anthraquinone (AQ code number), such as AQ8, AQ9 and AQ10, can trigger cytochrome c release without caspase activation and retain their ability to induce apoptosis in multidrug-resistant (MDR) tumor cells, fluorescent probes of transmembrane potential have been used to determine whether these anti-tumor compounds might directly target mitochondria in cell and cell-free systems to cause the collapse of mitochondrial membrane potential (/Deltapsim) that is linked to permeability transition pore (PTP) opening. "
11/01/2007 - "Synthetic analogs of 1,4-anthraquinone (AQ code number), which block nucleoside transport, decrease DNA, RNA and protein syntheses, trigger cytochrome c release without caspase activation, induce apoptotic DNA fragmentation and inhibit the proliferation of wild-type and multidrug resistant tumor cells in the nM range in vitro, rapidly cause the collapse of mitochondrial transmembrane potential in cell and cell-free systems. "
|1.||Cytochromes c (Cytochrome c)
|2.||RNA (Ribonucleic Acid)
|3.||Fluorescent Dyes (Fluorescent Probes)
|4.||DNA (Deoxyribonucleic Acid)