|1.||Belousov, Andrei B: 3 articles (01/2015 - 01/2012)|
|2.||Voss, Logan J: 3 articles (11/2010 - 01/2010)|
|3.||Sleigh, James W: 3 articles (11/2010 - 01/2010)|
|4.||Denisova, Janna V: 2 articles (01/2015 - 01/2012)|
|5.||Fontes, Joseph D: 2 articles (01/2015 - 01/2012)|
|6.||Weiler, Reto: 2 articles (01/2013 - 05/2005)|
|7.||Sayyah, Mohammad: 2 articles (01/2013 - 04/2010)|
|8.||Paul, David L: 2 articles (03/2010 - 11/2002)|
|9.||Bloomfield, Stewart A: 2 articles (03/2010 - 11/2002)|
|10.||Zhong, Wei: 1 article (01/2016)|
01/01/2013 - "There are abundant studies indicating that blocking gap junctions (GJs) containing connexin 36 (Cx36) inhibit seizures. "
01/01/2010 - "The aim of this study was to clarify the role of connexin36 (Cx36) gap junctions in neocortical seizures. "
07/01/2012 - "Changes in the connexin 36 gene and protein were accompanied by seizures. "
01/01/2010 - "Connexin36 gap junction blockade is ineffective at reducing seizure-like event activity in neocortical mouse slices."
08/11/2006 - "Since connexin36 expression levels change after seizures, we examined the expression of both pannexin1 and connexin36 in cerebral cortex, hippocampus, cerebellum and brain stem at different time intervals (2, 4 and 8 h) after i.p. "
07/01/2012 - "This review discusses the experimental and clinical studies those show the expression of connexin 36 in the central nervous system and the possible role of connexin 36 in epileptic seizure. "
07/01/2012 - "Original articles and reviews were selected if they were related to the expression of connexin 36 in the central nervous system and its role in epilepsy. "
07/01/2012 - "Expression of connexin 36 in central nervous system and its role in epileptic seizure."
08/11/2006 - "This finding suggested that pannexin1 and connexin36 are involved in different functional roles or that they are expressed in different cell types and that only those expressing the Cx36 are induced to apoptosis by epileptic seizures."
07/01/2012 - "A better understanding of the role of connexin 36 in seizure activity may contribute to the development of new therapeutic approaches to treating epilepsy."
|3.||Ganglion Cysts (Ganglion)
11/14/2002 - "A new study in this issue of Neuron shows that all pathways from rods to ON-type ganglion cells require connexin36. "
04/01/2011 - "AAV2 vector incorporating the neuronal promoter human connexin 36 (hCx36) transduced ganglion cells within a dense annulus around the fovea center, whereas AAV2 containing the ubiquitous promoter hybrid cytomegalovirus (CMV) enhancer/chicken-β-actin (CBA) transduced both Müller and ganglion cells in a dense circular disc centered on the fovea. "
03/15/2010 - "Both connexin36 (Cx36) and connexin45 (Cx45) subunits are widely distributed in the inner plexiform layer (IPL) and therefore are likely contribute to gap junctions formed by a number of ganglion cell subtypes. "
03/15/2010 - "Connexin36 is required for gap junctional coupling of most ganglion cell subtypes in the mouse retina."
05/09/2005 - "Connexin36 mediates gap junctional coupling of alpha-ganglion cells in mouse retina."
09/01/2012 - "Specifically, we found that activation of group II mGluRs increases background levels of neuronal gap junction coupling and expression of connexin 36 (Cx36; neuronal gap junction protein), whereas inactivation of group II mGluRs prevents the ischemia-mediated increases in the coupling and Cx36 expression. "
01/11/2012 - "Specifically, using electrotonic coupling, Western blots, and siRNA in the mouse somatosensory cortex in vivo and in vitro, we demonstrate that activation of group II mGluRs increases background levels of neuronal gap junction coupling and expression of connexin 36 (Cx36) (neuronal gap junction protein), and inactivation of group II mGluRs prevents the ischemia-mediated increases in the coupling and Cx36 expression. "
01/01/2015 - "Pharmacological blockade or genetic knockout of neuronal connexin 36 (Cx36)-containing gap junctions reduces neuronal death caused by ischemia, traumatic brain injury and NMDA receptor (NMDAR)-mediated excitotoxicity. "
|3.||Proteins (Proteins, Gene)
|6.||Small Interfering RNA (siRNA)
|7.||4-Aminopyridine (4 Aminopyridine)
|8.||Messenger RNA (mRNA)
|9.||Glutamic Acid (Glutamate)
|10.||Neurotransmitter Agents (Neurotransmitter)