|1.||Iso-o, Naoyuki: 1 article (04/2005)|
|2.||Mori, Ichiro: 1 article (04/2005)|
|3.||Nagai, Ryozo: 1 article (04/2005)|
|4.||Sata, Masataka: 1 article (04/2005)|
|5.||Aizawa, Toru: 1 article (04/2005)|
|6.||Ohno, Minoru: 1 article (04/2005)|
|7.||Noiri, Eisei: 1 article (04/2005)|
|8.||Saito, Kan: 1 article (04/2005)|
|9.||Ishizaka, Nobukazu: 1 article (04/2005)|
|10.||Hashimoto, Y: 1 article (03/2001)|
03/01/2001 - "In another series of experiments, the control (n = 5) and T-0970 (n = 5) groups were killed 4 h after reperfusion following 30 min coronary occlusion and DNA fragmentation in myocytes was assessed using in situ dUTP nick end-labelling (TUNEL) at the light microscopic level. "
03/01/2001 - "2. T-0970 scavenged hydroxyl radicals generated in the myocardium during reperfusion, as assessed by using a microdialysis technique and HPLC in an in vivo model with 30 min coronary occlusion and 30 min reperfusion in rabbits. "
04/01/2005 - "T-0970 and deferoxamine suppressed the upregulation of TGF-beta1 mRNA and reduced the extent of cardiac fibrosis in the heart of rats treated with angiotensin II. These agents blocked the angiotensin II-induced upregulation of heme oxygenase-1, a potent oxidative and cellular stress-responsive gene, but they did not significantly affect systolic blood pressure or plasma levels of aldosterone. "
|1.||Transforming Growth Factor beta1 (TGF beta 1)
|4.||Messenger RNA (mRNA)
|8.||tranilast (N 5')