|1.||Katunuma, Nobuhiko: 2 articles (06/2014 - 11/2009)|
|2.||Zhang, Hui-Ling: 1 article (06/2014)|
|3.||Ni, Yong: 1 article (06/2014)|
|4.||Xu, Min: 1 article (06/2014)|
|5.||Rong, Jia-Guo: 1 article (06/2014)|
|6.||Yang, Lei: 1 article (06/2014)|
|7.||Luo, Yu: 1 article (06/2014)|
|8.||Ishidoh, Kazumi: 1 article (06/2014)|
|9.||Gu, Wei-Wei: 1 article (06/2014)|
|10.||Li, Zhong-Sheng: 1 article (06/2014)|
08/01/2001 - "Taken together, these findings suggest that treatment of host mice with CLIK148 affects the processing of SLA in APC, resulting in the potentiation of T (h) 2-type immune responses and thus leading to exacerbation of the infection. "
11/25/2009 - "The CA-074Me treatment of NIH3T3 cells inhibited cathepsin L activity, and a cathepsin L specific inhibitor, CLIK148, attenuated the Eco-MLV vector infection. "
11/25/2009 - "The CA-074Me treatment inhibited the Eco-MLV infection in human cells expressing the exogenous mouse ecotropic receptor and endogenous cathepsins B and L, but the CLIK148 treatment did not, showing that only the cathepsin L suppression by CLIK148 is not enough to prevent the Eco-MLV infection in cells expressing both of cathepsins B and L, and CA-074Me inhibits the Eco-MLV infection by suppressing both of cathepsins B and L. "
08/01/2001 - "Interestingly, treatment of BALB/c mice with CLIK148 exacerbated the infection by enhancing the development of SLA-specific T (h) 2-type response such as production of IL-4 and generation of T (h) 2-dependent specific IgE/IgG1 antibodies. "
|1.||CA 074 methyl ester
|3.||Caspase 3 (Caspase-3)
|4.||Interleukin-4 (Interleukin 4)
|5.||Immunoglobulin G (IgG)
|6.||Immunoglobulin E (IgE)