|1.||Hussain, Afthab: 1 article (04/2014)|
|2.||Maddock, Helen L: 1 article (04/2014)|
|3.||Harvey, Kate L: 1 article (04/2014)|
|4.||Gao, Qun: 1 article (09/2008)|
|5.||Wolin, Michael S: 1 article (09/2008)|
|6.||Kisaalita, W S: 1 article (12/2006)|
|7.||Zhao, Y-P: 1 article (12/2006)|
|8.||Wu, Z-Z: 1 article (12/2006)|
|9.||Grobler, Erika: 1 article (02/2006)|
|10.||Van der Schyf, Cornelis J: 1 article (02/2006)|
06/01/1996 - "Digital imaging (confocal microscopy) and a slow potentiometric dye (tetramethylrhodamine methyl ester) were used to assess the resting membrane potential (V m) of murine neuroblastoma cells (N1E-115). "
02/15/2006 - "A series of pentacycloundecylamine derivatives were synthesized and their influence on the transmembrane potential of human SH-SY5Y neuroblastoma cells was evaluated using laser scanning confocal microscopy in combination with the potentiometric dye tetramethylrhodamine methyl ester. "
08/01/1996 - "A voltage-sensitive probe, tetramethylrhodamine methyl ester (TMRM) and digital imaging (confocal microscopy) were used to quantify differential membrane potential alteration in neuronal (murine neuroblastoma, N1E-115) and non-neuronal (human epithelial-like, HeLa) cells, after 1 and 24 hr of exposure to a knownin vivo neurotoxic agent, acetylsalicylic acid (ASA), as a first step in substantiating the relevance of alteration in resting membrane potential (V(m)) as a neurotoxic endpoint. "
12/15/2006 - "Resting membrane potentials and voltage-gated calcium channel (VGCC) function of SH-SY5Y human neuroblastoma cells cultured on the microbead array versus collagen-coated flat (2-D) substrates were evaluated by confocal microscopy with a potentiometric dye, tetramethylrhodamine methyl ester, and a calcium fluorescent indicator, Calcium Green-1. "
04/01/2014 - "Mitochondrial-associated responses were detected by tetramethylrhodamine methyl ester fluorescence and myocyte contracture. "
08/01/2004 - "Adult rat myocytes were loaded with the fluorescent probe tetramethylrhodamine methyl ester, which generates oxidative stress on laser illumination, thus inducing the mPT (indicated by collapse of the mitochondrial membrane potential) and ATP depletion, seen as rigor contracture. "
09/01/2008 - "Hypoxia appeared to increase mitochondrial and decrease cytosolic-nuclear superoxide under conditions associated with increased cytosolic NADH (lactate/pyruvate), mitochondrial NAD(P)H, and hyperpolarization of mitochondria detected by tetramethylrhodamine methyl-ester perchlorate fluorescence. "
|2.||Pyruvic Acid (Pyruvate)
|3.||Calcium Channels (Calcium Channel)
|8.||Aspirin (Acetylsalicylic Acid)
|9.||Adenosine Triphosphate (ATP)