adenovirus E4orf4 protein

05/07/2015 - "Mechanisms of cancer cell killing by the adenovirus E4orf4 protein."
04/01/2015 - "The adenovirus E4orf4 protein expressed at high levels kills cancer cells but not normal human primary cells. "
04/01/2015 - "Analysis by live imaging of effects of the adenovirus E4orf4 protein on passage through mitosis of H1299 tumor cells."
01/01/2014 - "Induction of cancer-specific cell death by the adenovirus E4orf4 protein."
01/01/2013 - "Adenovirus E4orf4 protein induces the death of human cancer cells and Saccharomyces cerevisiae. "

01/01/2014 - "The adenovirus E4orf4 protein is a multifunctional viral regulator that contributes to temporal regulation of the progression of viral infection. "
01/01/2020 - "Biology of the adenovirus E4orf4 protein: from virus infection to cancer cell death."
06/01/2014 - "The adenovirus E4orf4 protein contributes to regulation of the progression of virus infection from the early to the late phase, and when expressed alone, it induces a unique caspase-independent programmed cell death which is more efficient in cancer cells than in normal cells. "
05/10/2013 - "The adenovirus E4orf4 protein regulates the progression of viral infection and when expressed outside the context of the virus it induces nonclassical, cancer cell-specific apoptosis. "
05/07/2013 - "The adenovirus E4orf4 protein regulates the progression of viral infection, and when expressed alone in mammalian tissue culture cells it induces protein phosphatase 2A (PP2A)-B55- and Src-dependent cell death, which is more efficient in oncogene-transformed cells than in normal cells. "

12/01/2013 - "Adenovirus E4orf4 protein-induced death of p53-/- H1299 human cancer cells follows a G1 arrest of both tetraploid and diploid cells due to a failure to initiate DNA synthesis."

02/01/2009 - "Localization and importance of the adenovirus E4orf4 protein during lytic infection."

01/22/2009 - "The adenovirus E4orf4 protein induces growth arrest and mitotic catastrophe in H1299 human lung carcinoma cells."