|1.||Li, Hongliang: 6 articles (11/2015 - 10/2011)|
|2.||He, You-Wen: 6 articles (08/2012 - 01/2004)|
|3.||Zhang, Yan: 4 articles (07/2015 - 10/2011)|
|4.||Zhu, Li-Hua: 3 articles (11/2015 - 05/2014)|
|5.||Li, Hong: 3 articles (05/2008 - 01/2004)|
|6.||Zhang, Peng: 2 articles (11/2015 - 07/2015)|
|7.||Tian, Song: 2 articles (11/2015 - 07/2015)|
|8.||Zhang, Xiaodong: 2 articles (05/2014 - 09/2013)|
|9.||Jiang, Ding-Sheng: 2 articles (05/2014 - 08/2012)|
|10.||Deng, Shan: 2 articles (09/2013 - 08/2012)|
|1.||Cardiomegaly (Heart Hypertrophy)
08/01/2012 - "In this study, we investigated the role of mindin in cardiac hypertrophy using littermate Mindin knockout (Mindin ( -/- )) and wild-type (WT) mice. "
10/01/2011 - "This study was aimed to identify the effect of mindin on cardiac hypertrophy and the underlying mechanisms. "
10/01/2011 - "To further investigate the role of mindin in cardiac hypertrophy, we used cultured neonatal rat cardiomyocytes with gain and loss of mindin function and cardiac-specific Mindin-overexpressing transgenic (TG) mice. "
10/01/2011 - "Mindin is a secreted extracellular matrix protein, an integrin ligand, and an angiogenesis inhibitor, other examples of which are all key players in the progression of cardiac hypertrophy. "
10/01/2011 - "Cardiac-specific mindin overexpression attenuates cardiac hypertrophy via blocking AKT/GSK3β and TGF-β1-Smad signalling."
11/01/2015 - "Our previous studies revealed that Mindin is involved in the pathogenesis of ischemic stroke. "
09/01/2013 - "Mindin-deficient mice exhibited low expression levels of stroke-induced inflammatory mediators, an attenuated recruitment of inflammatory cells, and inhibited activation of NF-κB. "
09/01/2013 - "These data demonstrate that mindin is a critical mediator of ischemic brain injury in an experimental stroke model. "
09/01/2013 - "Mindin is a critical mediator of ischemic brain injury in an experimental stroke model."
05/01/2008 - "In this study, we examined the role of mindin in influenza virus infection. "
05/01/2008 - "We showed that mindin interacted with influenza virus particles directly and that mindin-deficient macrophages exhibited impaired activation after influenza virus infection in vitro. "
05/01/2008 - "We found that intranasal infection of mindin-deficient mice by influenza virus resulted in dramatically increased virus titers in the lung and intranasal cavity of mutant mice. "
05/01/2008 - "Together, these results demonstrate that mindin plays an essential role in the host innate immune response to influenza virus infection and suggest that mindin may be used as an immune-enhancing agent in influenza infection."
01/01/2014 - "gondii infection was observed in the mindin group compared with that of Freund's adjuvant and no-adjuvant groups. "
08/01/2012 - "Our data show that mindin, as an intrinsic cardioprotective factor, prevents maladaptive remodelling and the transition to heart failure by blocking AKT/GSK3β signalling."
10/01/2011 - "The present study demonstrates for the first time that mindin serves as a novel mediator that protects against cardiac hypertrophy and the transition to heart failure by blocking AKT/GSK3β and TGF-β1-Smad signalling."
08/01/2012 - "As an ECM protein, an integrin ligand, and an angiogenesis inhibitor, all of which are key players in cardiac hypertrophy, mindin is an attractive target for therapeutic intervention to treat or prevent cardiac hypertrophy and heart failure. "
09/01/2013 - "Mindin is an ECM protein that plays important roles in regulating inflammation, angiogenesis and neuronal outgrowth. "
03/07/2010 - "To investigate the regulation of mindin expression and the signaling pathway involved during inflammation. "
12/01/2005 - "In this report we show that mice lacking the ECM protein mindin exhibit severely impaired recruitment of neutrophils and macrophages in 4 different inflammation models. "
10/01/2011 - "After inhalation of ozone, mice had no detectible differences in cellular inflammation or total BALF protein dependent on mindin. "
03/07/2010 - "Mindin expression is upregulated during intestinal inflammation and may induce NF-kappaB promoter activation in a TLR-9 mediated manner."
|1.||Glycogen Synthase Kinases
|2.||NF-kappa B (NF-kB)
|3.||Transforming Growth Factors (Transforming Growth Factor)
|4.||Messenger RNA (mRNA)
|1.||Heterologous Transplantation (Xenotransplantation)