|1.||Tao, Yuan-Xiang: 3 articles (02/2011 - 05/2005)|
|2.||Shimamoto, Keiko: 2 articles (04/2014 - 07/2002)|
|3.||Rothstein, Jeffery D: 2 articles (02/2011 - 05/2005)|
|4.||Bae, Yong C: 1 article (03/2015)|
|5.||Ju, Jin S: 1 article (03/2015)|
|6.||Park, Ki D: 1 article (03/2015)|
|7.||Kim, Min J: 1 article (03/2015)|
|8.||Kim, Seong T: 1 article (03/2015)|
|9.||Ahn, Dong K: 1 article (03/2015)|
|10.||Mun, Jun H: 1 article (03/2015)|
01/01/2007 - "The ischemia-induced inward currents were also substantially inhibited by the glutamate transporter inhibitor TBOA (100 microM). "
06/01/2005 - "Likewise, WAY-855, did not significantly exacerbate neurotoxicity associated with simulated ischemia, whereas TBOA did. "
02/21/2003 - "The glutamate transporter inhibitor D,L-TBOA reduced ischemia-induced efflux throughout a 45-min incubation period, and enhanced efflux from control tissue. "
08/01/2003 - "The glutamate transporter inhibitor DL-TBOA prevented the ischemia-evoked efflux of [(3)H]D-aspartate by about 40%, indicating a carrier-mediated efflux. "
06/16/2000 - "Basal and ischemia-evoked amino acid, glucose and lactate efflux were compared in control versus DL-TBOA (100 microM; applied topically for 35 min prior to ischemia) animals. "
02/01/2011 - "Intrathecal TBOA also attenuated CFA-induced thermal hyperalgesia at 6 h after CFA injection. "
03/03/2015 - "Intracisternal administration of high dose TBOA (10 μg) produced thermal hyperalgesia in naïve rats but significantly attenuated the thermal hyperalgesia in rats that had been pretreated with interleukin (IL)-1β or Complete Freund's Adjuvant (CFA). "
03/23/2001 - "On the other hand, the glutamate transporter blocker DL-threo-beta-benzyloxyaspartate (DL-TBOA) blocked all the allodynia induced by PGE(2), PGF(2 alpha), NMDA, and AMPA. "
09/15/2010 - "In contrast, cells exposed to either chemical anoxia or high KCl elicited a marked release of d-[(3)H] aspartate, and the release was inhibited by TBOA and DHK, implying the contribution of glutamate transporter reversal. "
05/01/2007 - "Neither 6-cyano-7-nitroquinoxaline-2,3-dione disodium, DL: -threo-beta-benzyloxyaspartate, nifedipine, nor bepridil inhibited the hypoxia-induced [Ca(2+)](i) increase. "
|2.||Glutamic Acid (Glutamate)
|3.||Aspartic Acid (Aspartate)
|4.||Amino Acid Transport System X-AG (Glutamate Transporter)
|6.||D-Aspartic Acid (D Aspartate)
|8.||6-Cyano-7-nitroquinoxaline-2,3-dione (6 Cyano 7 nitroquinoxaline 2,3 dione)
|9.||alpha- Amino- 3- hydroxy- 5- methyl- 4- isoxazolepropionic Acid (AMPA)
|10.||Metabotropic Glutamate Receptors (Metabotropic Glutamate Receptor)