|1.||Oh, Ki-Bong: 2 articles (04/2015 - 05/2010)|
|2.||Shin, Jongheon: 2 articles (04/2015 - 05/2010)|
|3.||Daidone, Giuseppe: 1 article (12/2015)|
|4.||Raimondi, Maria Valeria: 1 article (12/2015)|
|5.||Schillaci, Domenico: 1 article (12/2015)|
|6.||Cascioferro, Stella: 1 article (12/2015)|
|7.||Raffa, Demetrio: 1 article (12/2015)|
|8.||Maggio, Benedetta: 1 article (12/2015)|
|9.||Yang, Hyeong-Cheol: 1 article (04/2015)|
|10.||Lee, So-Hyoung: 1 article (04/2015)|
|2.||Gram-Positive Bacterial Infections
10/01/2014 - "Sortase A (srtA) is the key enzyme that anchors pre-mature cell wall-attached proteins to peptidoglycan and it can affect the correct positioning of surface proteins, as well as the course of Gram-positive bacterial infection, thereby making it a potential target in the study of virulence factors and disease control. "
01/01/2015 - "Sortase A (SrtA) has attracted great interest as a potential drug target to treat infections caused by S. "
08/01/2011 - "Here, we first show using deletion analysis and in vivo infection, that InlK is a bona fide virulence factor, poorly expressed in vitro and well expressed in vivo, and that it is anchored to the bacterial surface by sortase A. "
05/28/2010 - "The rate of kidney infection was significantly reduced in the mice treated with sortase A knock-out S. "
01/01/2015 - "The therapeutic effect of chlorogenic acid against Staphylococcus aureus infection through sortase A inhibition."
03/18/2008 - "suis mutant strain (SRTDeltaA) lacking the sortase A gene (srtA) to interact with host cells and extracellular matrix (ECM) proteins, as well as its virulence in a mouse infection model. "
|4.||Dental Caries (White Spot)
04/01/2015 - "Based upon the results of combined spectroscopic methods, the structure of new compound (2) was determined to be maltol-3-O-(4'-O-cis-p-coumaroyl-6'-O-(3-hydroxy-3-methylglutaroyl))-β-glucopyranoside, an isomer of 1. These compounds strongly inhibited the action of sortase A (SrtA) from Streptococcus mutans, a primary etiologic agent of human dental caries. "
01/01/2014 - "mutans) is the main etiological agent of dental caries, and adheres to the tooth surface through the sortase A (SrtA)-mediated cell wall-anchored protein Pac. "
|5.||Infectious Arthritis (Septic Arthritis)
07/01/2003 - "Together these results indicate that sortase B (srtB) plays a contributing role during the pathogenesis of staphylococcal infections, whereas sortase A (srtA) is an essential virulence factor for the establishment of septic arthritis."
02/01/2008 - "The Staphylococcus aureus cell surface protein clumping factor A (ClfA) and the enzyme sortase A (SrtA), which attach surface proteins to the cell wall, have both been shown to be virulence factors in models of septic arthritis and sepsis. "
|2.||Proteins (Proteins, Gene)
|3.||Virulence Factors (Pathogenicity Factors)
|4.||Membrane Proteins (Integral Membrane Proteins)
|7.||Messenger RNA (mRNA)