|1.||Martin, X: 2 articles (11/2001 - 03/2000)|
|2.||Da Silva, M: 2 articles (11/2001 - 03/2000)|
|3.||Tiollier, J: 2 articles (11/2001 - 03/2000)|
|4.||Baldwin, W M: 1 article (05/2011)|
|5.||Fairchild, R L: 1 article (05/2011)|
|6.||Hattori, Y: 1 article (05/2011)|
|7.||Tanabe, K: 1 article (05/2011)|
|8.||Ishii, D: 1 article (05/2011)|
|9.||Schenk, A D: 1 article (05/2011)|
|10.||Setoguchi, K: 1 article (05/2011)|
11/01/2001 - "We established a primate model to investigate the effects of the antileukocyte function associated antigen 1 (CD 11a) mAb odulimomab (Imtix-Sangstad, Lyon, France) for preventing renal ischemia-reperfusion injury. "
07/01/1999 - "The goal of this study is to determine the effects of ex vivo hyperbaric pressure administration of AS-ICAM-1 ODN and systemic anti-LFA-1 mAb treatment on reperfusion injury in the rat cardiac allograft model. "
07/01/1999 - "Ex vivo pressure mediated delivery of AS-ICAM-1 ODN decreases ICAM-1 protein expression, reduces reperfusion injury in rodent cardiac allografts, and is more effective than anti-LFA-1 mAb treatment alone."
03/10/2005 - "Anti-LFA-1 mAb or anti-VLA-4 mAb alone had no effect on adhesion or transendothelial cancer cell migration, but were able to inhibit both of these functions when added together. "
01/01/1992 - "Tumor progression in anti-LFA-1 MAb-injected mice is explained mostly by blockage of CTL lytic activity at the tumor site; in mice receiving i.p. "
01/01/1992 - "Local and systemic treatment with high doses of anti-LFA-1 monoclonal antibody led to tumor progression by blocking CTL interaction with tumor cells without interfering with CTL generation and localization in the tumor mass."
12/01/1990 - "Moreover, the autotumor cytotoxicity was inhibited by anti-HLA class I but not by anti-CD1c or anti-LFA-1 mAb, suggesting that killing of the autologous tumor cells and non-major histocompatibility complex-restricted cytotoxicity are mediated by different mechanisms."
09/15/1990 - "Anti-LFA-1 MAb added after conjugate formation still inhibited lysis of both ICAM-1+or- tumor cells. "
04/10/1999 - "Enhanced neutralization by anti-gp120 MAbs was observed if the anti-LFA-1 MAb was present during the initial 24 hr only, if added 24 hr after infection, or if present throughout the culture period. "
08/01/1998 - "Anti-LFA-1 monoclonal antibody in renal transplantation: renal function, infections, and other complications."
10/01/1988 - "Anti-LFA-1 mAb appeared to be more potent at inhibiting cutaneous GVHD rather than delayed-type hypersensitivity responses, whereas anti-L3T4 mAb inhibited equally both the responses. "
10/01/1988 - "Prevention of cutaneous GVHD was achieved with local administration of either anti-L3T4 or anti-LFA-1 mAb, but not with anti-I-A mAb, while delayed-type hypersensitivity responses were inhibited by all the mAb. These results indicate that an I-A Ag-independent mechanism may be also operative in the development of the cutaneous GVHD, unlike the delayed-type hypersensitivity responses. "
07/15/1997 - "However, local expression of IgM, C3 and T helper 1 cytokines, serum antibodies of IgG and IgM, and delayed-type hypersensitivity were suppressed in the anti-LFA-1 mAb- plus FK506-treated group. "
|5.||Allergic Contact Dermatitis
09/01/1994 - "To investigate the importance of one of the CAMs, lymphocyte function-associated antigen-1 (LFA-1) in allergic contact dermatitis (ACD), a specific anti-LFA-1 monoclonal antibody was injected into the ears of mice after sensitization but prior to challenge with dinitrofluorobenzene (DNFB). "
|2.||Intercellular Adhesion Molecule-1 (Intercellular Adhesion Molecule 1)
|3.||Lymphocyte Function-Associated Antigen-1 (LFA-1)
|4.||Immunoglobulin G (IgG)
|1.||Homologous Transplantation (Allograft)
|4.||Transplantation (Transplant Recipients)