|1.||Singh, Sukh Mahendra: 5 articles (12/2013 - 11/2007)|
|2.||Salimath, Bharathi P: 3 articles (08/2007 - 05/2005)|
|3.||Singh, Vivek: 2 articles (12/2013 - 11/2007)|
|4.||Vishvakarma, Naveen Kumar: 2 articles (12/2013 - 10/2011)|
|5.||Jiang, Ding-Sheng: 1 article (04/2015)|
|6.||Li, Hongliang: 1 article (04/2015)|
|7.||Huang, Kun: 1 article (04/2015)|
|8.||Huang, Kai: 1 article (04/2015)|
|9.||Zhang, Xiao-Dong: 1 article (04/2015)|
|10.||Gao, Lu: 1 article (04/2015)|
09/01/2011 - "DNaseI activity was dramatically reduced only in kidneys of mice with sever nephritis and was the only nuclease that was down-regulated, whereas six other nucleases (DNaseII1 to 3, caspase-activated DNase, Dnase2a, and endonuclease G) were approximately normally expressed in kidneys, liver, and spleen. "
04/01/2011 - "Confocal microscopy analysis indicates that granzyme B treatment of tumor cells induced an early translocation of endonuclease caspase-activated DNase. "
04/01/2010 - "Summarily, these results suggest a cytotoxic stress (resembling DNA damage) response of tumor cells to Dz13, which induces apoptosis via the activation of inhibitor of caspase-activated deoxyribonuclease and protein kinase C delta. "
09/29/2005 - "Caspase-activated DNase (CAD), whose gene (hCAD) is mapped at chromosome 1p36, a region frequently displaying hemizygote deletion in many human cancers, is the key enzyme for nucleosome fragmentation during apoptosis. "
03/01/2006 - "indica is mediated by NF-kappaB and Caspase activated DNase in ascites tumor model."
01/27/2003 - "The gene of caspase-activated DNase (CAD), the key enzyme for nucleosome cleavage during apoptosis, is mapped at chromosome 1p36, a region usually associated with hemizygous deletions in human cancers, particularly in hepatoma (HCC). "
01/01/1998 - "A caspase-activated deoxyribonuclease (CAD) and its inhibitor (ICAD) have now been identified in the cytoplasmic fraction of mouse lymphoma cells. "
11/01/2007 - "An increase in the expression of heat shock protein (Hsp70), vascular endothelial growth factor, interleukin-2 receptor and interleukin-2 proteins along with an inhibition in the expression of caspase-activated DNase and p53 proteins was observed during the late tumor-bearing stage and also in the Dalton's lymphoma cells when cultured in vitro at a higher cell density. "
01/01/2002 - "These results suggest that caspase-3-dependent caspase-activated deoxyribonuclease activity plays an important role in mediating DNA fragmentation after focal ischemia."
12/01/2001 - "Here, we focused on two terminal cell death effectors; caspase-activated DNase (CAD) and lysosomal enzyme DNase II, in the monkey CA1 sector undergoing 18 min ischemia. "
|5.||Huntington Disease (Huntington's Disease)
10/03/2005 - "Age at onset of Huntington disease is not modulated by the R72P variation in TP53 and the R196K variation in the gene coding for the human caspase activated DNase (hCAD)."
02/10/2005 - "Modulation of age at onset of Huntington disease patients by variations in TP53 and human caspase activated DNase (hCAD) genes."
|2.||Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF)
|3.||Macrophage Colony-Stimulating Factor
|4.||Caspase 3 (Caspase-3)
|5.||DNA (Deoxyribonucleic Acid)
|6.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|7.||Proteins (Proteins, Gene)
|8.||NF-kappa B (NF-kB)