|1.||Spear, P G: 1 article (10/2007)|
|2.||Ch'ng, T H: 1 article (10/2007)|
|3.||Struyf, F: 1 article (10/2007)|
|4.||Enquist, L W: 1 article (10/2007)|
|5.||Nauwynck, Hans J: 1 article (06/2005)|
|6.||Ficinska, Jolanta: 1 article (06/2005)|
|7.||Favoreel, Herman W: 1 article (06/2005)|
|8.||Van Minnebruggen, Geert: 1 article (06/2005)|
|9.||Bienkowska-Szewczyk, Krystyna: 1 article (06/2005)|
|10.||Tomishima, Mark J: 1 article (08/2002)|
10/01/2007 - "We also found that unlike PRV gD, HSV-1 gD is required for neuron-to-cell spread of infection. "
08/01/2002 - "We observed that both wild-type virus and a PRV gD null mutant are capable of spreading from axons to closely apposed nonneuronal cells within the rat optic nerve after intravitreal infection. "
11/01/2001 - "In contrast, whereas wild-type PrV-infected pigs showed clinical signs and histological and histopathological findings typical of PrV infection, no signs of disease were observed after infection with PrV-gD(-) Pass. "
11/01/2001 - "To analyze whether this alteration in receptor usage in vitro influences infection in vivo, the model host mouse and the natural host pig were intranasally infected with PrV-gD(-) Pass and were compared to animals infected by wild-type PrV. "
11/01/2001 - "However, in the presence of compensatory mutations, infection can also occur in the absence of gD, as evidenced by the isolation in cell culture of an infectious gD-negative PrV mutant (PrV-gD(-) Pass) (J. "
|1.||Polyethylene Glycols (Polyethylene Glycol)
|3.||DNA (Deoxyribonucleic Acid)