|1.||Zhang, Yanling: 3 articles (11/2014 - 07/2007)|
|2.||Weisman, Lois S: 3 articles (11/2014 - 07/2007)|
|3.||Ho, Cheuk Y: 2 articles (04/2015 - 03/2013)|
|4.||Botelho, Roberto J: 2 articles (04/2015 - 03/2013)|
|5.||McCartney, Amber J: 2 articles (11/2014 - 01/2014)|
|6.||Tronchère, Hélène: 2 articles (11/2006 - 02/2004)|
|7.||Laporte, Jocelyn: 2 articles (11/2006 - 02/2004)|
|8.||Payrastre, Bernard: 2 articles (11/2006 - 02/2004)|
|9.||Mandel, Jean-Louis: 2 articles (11/2006 - 02/2004)|
|10.||Slochower, David R: 1 article (05/2015)|
|1.||Neurodegenerative Diseases (Neurodegenerative Disease)
03/29/2013 - "Further highlighting its importance, PtdIns(3,5)P2 misregulation leads to the development of neurodegenerative diseases like Charcot-Marie-Tooth disease. "
01/01/2014 - "Recent studies of the low abundant signaling lipid, phosphatidylinositol 3,5-bisphosphate (PI(3,5)P2 ), reveal an intriguingly diverse list of downstream pathways, the intertwined relationship between PI(3,5)P2 and PI5P, as well as links to neurodegenerative diseases. "
10/01/2014 - "Thus, our studies suggest that the parasite PVM is able to fuse with host late endocytic vesicles in a PI(3,5)P2 -dependent manner, allowing the exchange of material between the host and the parasite, which is essential for successful infection. "
10/01/2014 - "Host PI(3,5)P2 activity is required for Plasmodium berghei growth during liver stage infection."
04/10/2015 - "Although the steady-state vacuolar pH is not affected by PtdIns(3,5)P2 depletion, it may have a role in stabilizing the vacuolar pH during salt shock. "
03/29/2013 - "Finally, we show that cells expressing monomeric Vac14 mutants have enlarged vacuoles that do not fragment after hyperosmotic shock, which indicates that PtdIns(3,5)P2 levels are greatly abated. "
01/21/2009 - "But how cells regulate PtdIns(3,5)P2 synthesis--for example, in response to hyperosmotic shock--remains unexplained. "
|4.||Congenital Structural Myopathies (Centronuclear Myopathy)
11/01/2006 - "A novel PtdIns3P and PtdIns(3,5)P2 phosphatase with an inactivating variant in centronuclear myopathy."
02/20/2004 - "These data demonstrate for the first time a role for MTM1 in the production of PtdIns(5)P in mammalian cells, suggesting that the lack of transformation of phosphatidylinositol 3,5-bisphosphate into PtdIns(5)P might be an important component in the etiology of myotubular myopathy."
|3.||Phosphatidylinositol 4,5-Diphosphate (Phosphatidylinositol 4,5 Diphosphate)