|1.||Furuyama, Kazumichi: 5 articles (01/2010 - 07/2004)|
|2.||Takeda, Kazuhisa: 5 articles (01/2010 - 07/2004)|
|3.||Shibahara, Shigeki: 5 articles (01/2010 - 07/2004)|
|4.||Regan, Raymond F: 4 articles (01/2014 - 11/2005)|
|5.||Ishikawa, Kazunobu: 4 articles (01/2010 - 07/2004)|
|6.||Date, Fumiko: 3 articles (01/2010 - 07/2004)|
|7.||Ogawa, Kazuhiro: 3 articles (07/2006 - 07/2004)|
|8.||Zhang, Yongzhao: 3 articles (07/2006 - 07/2004)|
|9.||Chen-Roetling, Jing: 2 articles (01/2014 - 03/2007)|
|10.||Szaflik, Jacek Pawel: 2 articles (03/2012 - 08/2011)|
|2.||Hirschsprung Disease (Hirschsprung's Disease)
01/01/2002 - "An experimental study on the expression of heme oxygenase-2 mRNA in Hirschsprung's disease."
01/01/2002 - "In order to investigate the relationship between the expression of heme oxygenase-2 (HO-2) mRNA and the pathogenesis of Hirschsprung's disease (HD), total ribonucleic acid (RNA) was extracted in the aganglionic and ganglionic segments of colon respectively from 15 cases of HD. "
03/26/2010 - "Enhanced translation of heme oxygenase-2 preserves human endothelial cell viability during hypoxia."
07/31/2009 - "Moreover, using C57BL/6 mice lacking heme oxygenase-2 (HO-2(-/-)), a model of chronic hypoxemia, we showed that the expression level of L-PGDS protein was twofold higher in the heart than that of wild-type mouse. "
01/01/2009 - "We have shown previously that the cardiorespiratory regions of the rostral ventrolateral medulla are excited by local hypoxia and that heme oxygenase-2 (HO-2) is expressed in the hypoxia-chemosensitive regions of the rostral ventrolateral medulla (RVLM), the respiratory pre-Bötzinger complex, and C1 sympathoexcitatory region. "
07/01/2006 - "Hypoxia reduces the expression of heme oxygenase-2 in various types of human cell lines. "
07/23/2004 - "Hypoxemia and blunted hypoxic ventilatory responses in mice lacking heme oxygenase-2."
|5.||Macular Degeneration (Age-Related Maculopathy)
08/01/2011 - "Lack of association between the c.544G>A polymorphism of the heme oxygenase-2 gene and age-related macular degeneration."
03/01/2012 - "On the other hand, oxidative stress has been implicated in the pathogenesis of age-related macular degeneration (AMD) and heme oxygenase-1 (HO-1), encoded by the HMOX1 gene and heme oxygenase-2 (HO-2), encoded by the HMOX2 gene are important markers of iron-related oxidative stress and its consequences. "
06/01/1999 - "The much higher heme oxygenase-1 and heme oxygenase-2 antigen levels in macular retinal pigment epithelium cells from younger individuals suggest that protective mechanisms against oxidation and, hence, presumably to the development of age-related macular degeneration, decrease with age."
06/01/1999 - "The high heme oxygenase-1 and heme oxygenase-2 lysosomal antigen levels in macular retinal pigment epithelium cells of eyes with neovascular age-related macular degeneration suggest that oxidative stress causes a pathologic upregulation of these enzymes. "
|2.||Messenger RNA (mRNA)
|5.||Endothelin-1 (Endothelin 1)
|6.||Nitric Oxide Synthase (NO Synthase)
|7.||Proteins (Proteins, Gene)
|8.||NADPH-Ferrihemoprotein Reductase (Cytochrome P450 Reductase)