|1.||Civeira, Maria Pilar: 1 article (08/2004)|
|2.||Riezu-Boj, Jose-Ignacio: 1 article (08/2004)|
|3.||Prieto, Jesús: 1 article (08/2004)|
|4.||Aldabe, Rafael: 1 article (08/2004)|
|5.||Guitart, Anunciata: 1 article (08/2004)|
|6.||Baixeras, Elena: 1 article (08/2004)|
|7.||Larrea, Esther: 1 article (08/2004)|
|8.||Carr, Daniel J J: 1 article (08/2003)|
|9.||Silverman, Robert: 1 article (08/2003)|
|10.||Al-khatib, Khaldun: 1 article (08/2003)|
08/01/2003 - "Cells treated with mouse IFN-beta consistently showed the greatest resistance to HSV-1 infection in comparison to cells treated with IFN-alpha1, IFN-alpha4, IFN-alpha5, IFN-alpha6, or IFN-alpha9. "
03/01/2001 - "In summary, our results indicate that virus-induced activation of the IFN system in human PBMC is associated with selective expression of individual IFN-alpha subtypes, IFN-alpha5 being the specific subtype induced in PBMC from patients with chronic HCV infection."
03/01/2001 - "In addition, we found that while natural HCV infection induced increased IFN-alpha5 expression in PBMC, in vitro infection of these cells with Sendai virus caused a raise in the expression of IFN-alpha8 in both patients and normal controls. "
08/01/2004 - "Hepatitis C virus (HCV) infection is associated with low IFN-alpha5 mRNA levels, possibly reflecting an escape mechanism of the virus. "
|3.||Chronic Hepatitis C
|4.||Hepatocellular Carcinoma (Hepatoma)
|1.||Messenger RNA (mRNA)