|1.||Xia, Jun: 1 article (01/2016)|
|2.||Zhang, Peifen: 1 article (01/2016)|
|3.||Yang, Chao: 1 article (01/2016)|
|4.||Zhou, Shili: 1 article (01/2016)|
|5.||Xu, Feng: 1 article (01/2016)|
|6.||Gao, Huixin: 1 article (01/2016)|
|7.||Wang, Yi: 1 article (01/2016)|
|8.||Li, Xiaobo: 1 article (01/2016)|
|9.||Zhang, Ping: 1 article (01/2016)|
|10.||Dolezel, Petr: 1 article (09/2015)|
05/01/1999 - "The prolonged residual elevation of cytosolic calcium characteristic for neurons of diabetic animals was also eliminated by carbonyl cyanide m-chlorophenylhydrazone, confirming the suggestion that such elevation is determined mainly by mitochondrial dysfunction, the latter being dependent on the level of hyperglycemia. "
06/01/2001 - "Thenoyltrifluoroacetone (TTFA), an inhibitor of mitochondrial electron transport chain complex II, and carbonyl cyanide m-chlorophenylhydrazone (CCCP), an uncoupler of oxidative phosphorylation, completely prevented the effects of hyperglycemia, suggesting that reactive oxygen species arise from the mitochondrial electron transport chain. "
02/01/2014 - "In this study, we report that increased mitochondrial PINK1 levels observed in human neuroblastoma SH-SY5Y cells after carbonyl cyanide m-chlorophelyhydrazone (CCCP) treatment were due to de novo protein synthesis, and not just increased stabilization of full length PINK1 (FL-PINK1). "
02/01/2000 - "Studies were conducted to determine the effects of bath application of the protonophores carbonyl cyanide m-chlorophenylhydrazone (CCCP) and carbonyl cyanide p-(trifluoromethoxy)-phenylhydrazone (FCCP) on membrane electrical characteristics of differentiated NG108-15 (neuroblastoma X glioma hybrid) cells. "
09/21/2014 - "A myocardial hypoxia environment was created using an oxygen consumption blocking reagent, carbonyl cyanide 4-(trifluoromethoxy)phenylhydrazone. "
11/01/2010 - "Respiratory responses of PC12 cells loaded with the conjugate, measured on a time-resolved fluorescent reader, showed significant cell deoxygenation in response to uncoupling by carbonyl cyanide 4-(trifluoromethoxy)phenylhydrazone and external hypoxia. "
09/03/2010 - "We showed here in a chemical hypoxia model of mitophagy induced by an uncoupler, carbonyl cyanide m-chlorophenylhydrazone (CCCP) that Parkin translocation resulted in mitochondrial ubiquitination and p62 recruitment to the mitochondria. "
07/01/2006 - "Mitochondrial uncoupling by carbonyl cyanide 4-(trifluoromethoxy) phenylhydrazone (FCCP, 1 microM) prior to hypoxia hastened the onset and shortened the duration of HSD. "
01/01/2005 - "During mitochondrial inhibition (due to exposure to carbonyl cyanide p-trifluromethoxyphenyl hydrazone (FCCP) and oligomycin), rises in [Ca2+]i (observed in Ca2+-free perfusate) evoked by hypoxia or by BK, were significantly enhanced, and hypoxia always evoked responses. "
|4.||Myocardial Ischemia (Ischemic Heart Diseases)
04/01/1999 - "Myocardial ischemia was simulated by the perfusion of metabolic inhibitors (3.3 mM amobarbital and 5 microM carbonyl cyanide m-chlorophenylhydrazone) with pH 6.6 and reperfusion was achieved by the washout of them with pH 7.4. "
04/01/2011 - "The purpose of this study was to investigate the roles of loss of oxidative phosphorylation and activation of the mitochondrial ATP-sensitive K+ channel and permeability transition pore in VF development during myocardial ischemia by using mitochondrial uncoupling agents (carbonyl cyanide m-chlorophenylhydrazone and 2,4-dinitrophenol) and channel blockers (5-hydroxydecanoate and cyclosporine A) at concentrations that have been demonstrated to block the intended targets selectively. "
|5.||Stomach Neoplasms (Stomach Cancer)
|2.||Carbonyl Cyanide m-Chlorophenyl Hydrazone (CCCP)
|3.||Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone (FCCP)
|4.||Adenosine Triphosphate (ATP)
|5.||Deoxyglucose (2 Deoxy D glucose)
|7.||2,4-Dinitrophenol (2,4 Dinitrophenol)