|1.||Yoshida, Masaru: 1 article (11/2010)|
|2.||Nishida, Masayuki: 1 article (11/2010)|
|3.||Mizushina, Yoshiyuki: 1 article (11/2010)|
|4.||Takikawa, Hirosato: 1 article (11/2010)|
|5.||Yoshida, Hiromi: 1 article (11/2010)|
|6.||Azuma, Takeshi: 1 article (11/2010)|
|7.||Maeda, Jun: 1 article (11/2010)|
11/01/2010 - "These results indicate that sulfobacin B is a potential chemotherapeutic agent for inflammation."
11/01/2010 - "In an in vivo mouse model of LPS-induced acute inflammation, the intraperitoneal injection of sulfobacin B to mice led to the suppression of serum TNF-α production. "
11/01/2010 - "Inhibitory effects of sulfobacin B on DNA polymerase and inflammation."
11/01/2010 - "However, sulfobacin B did not influence the activity of plant or prokaryotic pols, or that of the other DNA metabolic enzymes such as primase of pol α, RNA polymerase, polynucleotide kinase or deoxyribonuclease I. As we previously found a positive relationship between pol λ inhibition and anti-inflammation, we examined whether sulfobacin B could inhibit inflammatory responses. "
|1.||DNA Primase (Primase)
|2.||DNA-Directed RNA Polymerases (RNA Polymerase)
|3.||Polynucleotide 5'-Hydroxyl-Kinase (Kinase, Polynucleotide)
|5.||DNA (Deoxyribonucleic Acid)
|6.||Deoxyribonuclease I (Nickase)