|1.||Asadi, Abbas: 1 article (07/2015)|
|2.||Padulo, Johnny: 1 article (07/2015)|
|3.||Nikolaidis, Pantelis Theodoros: 1 article (07/2015)|
|4.||Chtourou, Hamdi: 1 article (07/2015)|
|5.||Calleja-González, Julio: 1 article (07/2015)|
|6.||Santos, Eduardo J A M: 1 article (07/2015)|
|7.||Zemkova, Erika: 1 article (07/2015)|
|8.||Attermann, Jorn: 1 article (06/2012)|
|9.||Wedderkopp, Niels: 1 article (06/2012)|
|10.||Moller, Merete: 1 article (06/2012)|
05/01/1995 - "On the other hand, the exposure of the cultures to U-18 during 15 hr hypoxia without subsequent reoxygenation also led to significant reduction of neuronal death compared with that observed without antioxidant. "
05/01/1995 - "The neuroprotective effects of both lipophylic U-18 and hydrophylic SOD on the cultures exposed to hypoxia/reoxygenation might reflect the damaging free radical overproduction in different morphofunctional compartments of the nerve cell."
05/01/1995 - "The application of U-18 to the cultures during 6-8 hr hypoxia followed by 16-18 hr reoxygenation in the absence of antioxidant significantly reduced neuronal death. "
09/01/1992 - "Morphological and biochemical quantitative analysis of neuronal damage showed that U-18 attenuates nerve cell death resultant from 6-7-hour hypoxia and subsequent 12-hour posthypoxic reoxygenation. "
01/01/1995 - "[The lipophilic antioxidant U-18 and superoxide dismutase prevent destruction of cultured hippocampal neurons during hypoxia and in the post-hypoxic period]."
|3.||Wounds and Injuries (Trauma)