|1.||Westcott, Jay Y: 1 article (10/2005)|
|2.||Huynh, Mai-Lan N: 1 article (10/2005)|
|3.||Kotaru, Chakradhar: 1 article (10/2005)|
|4.||Malcolm, Kenneth C: 1 article (10/2005)|
|5.||Tilstra, John A: 1 article (10/2005)|
|6.||Wenzel, Sally E: 1 article (10/2005)|
|7.||Fadok, Valerie A: 1 article (10/2005)|
|1.||Asthma (Bronchial Asthma)
10/15/2005 - "A decrease in AMphis LPS-responsiveness in severe asthma is manifested by defective apoptotic cell uptake and reduces secretion of inflammatory mediators. "
10/15/2005 - "Human bronchoalveolar lavage macrophages (AMphis) from normal control subjects and subjects with mild-moderate or severe asthma were examined in vitro for phagocytosis of apoptotic human T-cell line Jurkats and secretion of inflammatory mediators. "
10/15/2005 - "In contrast, AMphis from patients with severe asthma had defective LPS-stimulated uptake of apoptotic cells, with associated failure to induce PGE2 and 15-HETE. "
10/15/2005 - "AMphis from normal subjects and patients with mild-moderate asthma were able to phagocytose apoptotic cells in response to LPS, resulting in an induction of the antifibrotic and/or antiinflammatory eicosanoids, prostaglandin E2 (PGE2) and 15-hydroxyeicosatetraenoic acid (HETE). "
|3.||Hydroxyeicosatetraenoic Acids (HETE)