|1.||Kim, Eungbin: 1 article (11/2008)|
|2.||Yoo, Miyoun: 1 article (11/2008)|
|3.||Zylstra, Gerben J: 1 article (11/2008)|
|4.||Yon, Gyu Hwan: 1 article (11/2008)|
|5.||Choi, Sang-Un: 1 article (11/2008)|
|6.||Choi, Ki Young: 1 article (11/2008)|
|7.||Chae, Jong-Chan: 1 article (11/2008)|
|8.||Kim, Dockyu: 1 article (11/2008)|
11/07/2008 - "In addition, the cytotoxic effects of trisindoline were resistant to P-glycoprotein overexpression, one of the most common mechanisms of drug resistance in cancer cells, supporting its use to kill MDR cancer cells."
11/07/2008 - "While the cytotoxicity of etoposide, a common anticancer drug, was dramatically decreased in multidrug-resistant (MDR) cancer cells compared with treatment of parental cells, trisindoline was found to have similar cytotoxicity effects on both parental and MDR cell lines. "
11/07/2008 - "Bioactivity-guided fractionation of these indigo metabolites led to the isolation of trisindoline as the agent responsible for the observed in vitro cytotoxic activity against cancer cells. "
|2.||Etoposide (VP 16)