|1.||Urbaniak, Michael D: 3 articles (03/2014 - 06/2005)|
|2.||Ferguson, Michael A J: 3 articles (03/2014 - 06/2005)|
|3.||Crossman, Arthur: 2 articles (03/2014 - 06/2005)|
|4.||Swati, D: 1 article (12/2015)|
|5.||Rashmi, Mayank: 1 article (12/2015)|
|6.||Capes, Amy S: 1 article (03/2014)|
|7.||Thompson, Stephen: 1 article (03/2014)|
|8.||Gilbert, Ian H: 1 article (03/2014)|
|9.||O'Neill, Sandra: 1 article (03/2014)|
|10.||Abdelwahab, Nuha Z: 1 article (03/2012)|
|1.||Thymoma (Thymic Carcinoma)
04/16/2004 - "Previous studies of mouse thymoma cells showed that GlcNAc-PI de-N-acetylase activity is localized to the endoplasmic reticulum (ER) but enriched in a mitochondria-associated ER membrane (MAM) domain. "
04/16/2004 - "We show that human PIG-L is a type I membrane protein with a large cytoplasmic domain and that, unlike the result with mouse thymoma cells, both PIG-L and GlcNAc-PI-de-N-acetylase activity are uniformly distributed between ER and MAM in HeLa cells. "
|2.||African Trypanosomiasis (Nagana)
12/01/2015 - "In silico drug re-purposing against African sleeping sickness using GlcNAc-PI de-N-acetylase as an experimental target."
03/31/2014 - "The zinc-metalloenzyme GlcNAc-PI de-N-acetylase is essential for the biosynthesis of mature GPI anchors and has been genetically validated in the bloodstream form of Trypanosoma brucei, which causes African sleeping sickness. "
03/01/2012 - "Inhibitors incorporating zinc-binding groups target the GlcNAc-PI de-N-acetylase in Trypanosoma brucei, the causative agent of African sleeping sickness."
06/17/2005 - "Glycosylphosphatidylinositol biosynthesis is essential for Trypanosoma brucei, the causative agent of African sleeping sickness, and GlcNAc-PI de-N-acetylase has previously been validated as a drug target. "
|3.||Membrane Proteins (Integral Membrane Proteins)