|1.||Okabe, Masaru: 5 articles (05/2011 - 09/2004)|
|2.||Ikawa, Masahito: 4 articles (01/2011 - 09/2004)|
|3.||Yamaguchi, Ryo: 3 articles (07/2009 - 09/2004)|
|4.||Muro, Yuko: 2 articles (05/2011 - 07/2009)|
|5.||Inoue, Naokazu: 2 articles (01/2011 - 01/2007)|
|6.||Isotani, Ayako: 2 articles (07/2009 - 09/2004)|
|7.||Takumi, Kazuhiro: 1 article (07/2009)|
|8.||Tokuhiro, Keizo: 1 article (07/2009)|
|9.||Adham, Ibrahim: 1 article (07/2009)|
|10.||Kim, Dong Hoon: 1 article (10/2005)|
|1.||Male Infertility (Male Sterility)
01/01/2011 - "However, this is not likely, as there are already six different gene-disrupted mouse lines (Calmegin, Adam1a, Adam2, Adam3, Ace and Pgap1), all of which result in male sterility. "
09/01/2004 - "Loss of calmegin, a testis-specific putative chaperone protein of the endoplasmic reticulum, leads to male sterility because the sperm show defects in migration into the oviduct and do not bind to the zona pellucida. "
12/19/1997 - "The putative chaperone Calmegin is required for sperm fertility in mouse and the relevance of the gene to certain cases of human male infertility has been suggested. "
05/01/2011 - "Unexpectedly, sperm from 5 different gene-disrupted mouse lines (calmegin (Clgn), Adam1a, Adam2, Adam3, and Ace) all have defective zona-binding ability and oviduct-migrating ability. "
07/01/2009 - "Sperm from four different gene-disrupted mouse lines (calmegin [Clgn], Adam1a, Adam2, and Ace) are known to have defective zona-binding ability. "
01/01/2007 - "In the first half of this review we describe the interaction of ADAMs with calmegin and try to elucidate the relationship of these proteins to establish the zona binding ability. "
|1.||Complementary DNA (cDNA)
|2.||Messenger RNA (mRNA)
|3.||Proteins (Proteins, Gene)
|4.||trichostatin A (A 300)