|1.||Hardt, Wolf-Dietrich: 3 articles (01/2013 - 02/2004)|
|2.||Potter, Andrew A: 3 articles (03/2011 - 11/2005)|
|3.||Kitagawa, Masanobu: 2 articles (05/2012 - 08/2005)|
|4.||Giraud, Guillaume: 2 articles (01/2012 - 05/2009)|
|5.||Guyot, Boris: 2 articles (01/2012 - 05/2009)|
|6.||Morlé, François: 2 articles (01/2012 - 05/2009)|
|7.||Jepson, Mark A: 2 articles (07/2011 - 12/2008)|
|8.||Martinez-Argudo, Isabel: 2 articles (07/2011 - 12/2008)|
|9.||Lam, Po-King S: 2 articles (03/2011 - 07/2009)|
|10.||Berberov, Emil: 2 articles (03/2011 - 07/2009)|
12/01/2008 - "Recent studies have revealed a critical role for the SPI-1 effector SopB in 'directing traffic' at early stages of infection, allowing the bacteria to control SCV maturation by modulating its interaction with the endocytic system. "
01/01/2008 - "In our study, we highlight important differences in systemic and intestinal colonization levels between chick and murine serotype Typhimurium infections, and provide evidence that suggests that the role of SPI-1 may not be the same during colonization of both animal models."
01/01/2015 - "Sal147 was unable to invade HeLa cells and showed reduced mortality in Galleria mellonella infection model, in comparison to a SPI-1 harbouring S. "
01/01/2015 - "Salmonella enterica can cause intestinal or systemic infections in humans and animals mainly by the presence of pathogenicity islands SPI-1 and SPI-2, containing 39 and 44 genes, respectively. "
01/01/2014 - "During early infection (≤2 h), bacteria within both intact and compromised membrane-bound vacuoles were observed within the epithelium, with the majority expressing SPI-1. "
|2.||Salmonella Infections (Salmonellosis)
07/01/2011 - "The previously unrecognized strain heterogeneity in SPI-1 expression and invasiveness has important implications for studies of Salmonella infection."
11/01/2009 - "To cause infection, Salmonella enterica serovar Typhimurium uses type III secretion systems, which are encoded on two Salmonella pathogenicity islands, SPI-1 and SPI-2, the latter of which is thought to play a crucial role in bacterial proliferation in Salmonella-containing vacuoles (SCVs) after invading cells. "
11/01/2005 - "These data delineate novel phenotypes for SPI-1 and SPI-2 mutants in the intestinal phase of bovine and murine salmonellosis and provide working models to further investigate the effector contribution to these pathologies."
01/01/2009 - "In this study, we have constructed bacterial strains that contain a FLAG epitope inserted in frame to SPI-1 genes prgI, sipA, sipB, sopE2, spaO, and sptP, and investigated the expression of the tagged proteins both in vitro and in vivo during murine salmonellosis. "
01/01/2014 - "The possible correlation between attenuation of Salmonella infection in the chicken spleen and liver and reduction of Salmonella SPI-1 virulence gene expression in the chicken cecum by LAB isolates is a new observation. "
10/01/2011 - "SPI-1 genes are not essential for the diarrhea caused by S. "
09/01/1999 - "Mutations in SPI-1 (hilA and prgH) or aroA markedly reduced virulence and the severity of diarrhea. "
01/01/2008 - "A SPI-1 (invA) mutant caused cecal, but not colonic inflammation and did not cause diarrhea. "
04/01/2008 - "Recent studies of Salmonella enterica infection models have demonstrated that enterocolitis and diarrhea in mice and cows can occur independently of SPI-1. "
|1.||Proteins (Proteins, Gene)
|2.||Transcription Factors (Transcription Factor)
|3.||Serine Proteases (Serine Protease)
|4.||Messenger RNA (mRNA)
|6.||DNA-Activated Protein Kinase
|7.||Proto-Oncogene Proteins c-ets
|9.||Interleukin-18 (Interleukin 18)