|1.||Zolfaghari, Reza: 1 article (09/2012)|
|2.||Chen, Qiuyan: 1 article (09/2012)|
|3.||Ross, A Catharine: 1 article (09/2012)|
|4.||Kirschner, Ralf D: 1 article (06/2010)|
|5.||Müller, Gerd A: 1 article (06/2010)|
|6.||Engeland, Kurt: 1 article (06/2010)|
|7.||Rother, Karen: 1 article (06/2010)|
|8.||Guo, Xiaojia: 1 article (02/2002)|
|9.||Rinaldi, Christian: 1 article (02/2002)|
|10.||Screpanti, Isabella: 1 article (02/2002)|
06/01/2010 - "Taken together, the results suggest a potential role of the p53/p63-mediated retSDR1 activation in tumor suppression as well as in developmental processes."
06/01/2010 - "A tumor-derived p53 mutant is unable to activate retSDR1 transcription. "
02/15/2002 - "Our observations suggest that retSDR1 is a novel regulator of vitamin A metabolism and that its frequent deletion in NB cells bearing MYCN amplification could compromise the sensitivity of those cells to retinol, thereby contributing to cancer development and progression."
06/01/2010 - "The retinal dehydrogenase/reductase retSDR1/DHRS3 gene is activated by p53 and p63 but not by mutants derived from tumors or EEC/ADULT malformation syndromes."
|1.||Tretinoin (Retinoic Acid)
|4.||Vitamin A (Retinol)