|1.||Laast, Victoria A: 1 article (01/2007)|
|2.||Mankowski, Joseph L: 1 article (01/2007)|
|3.||Tarwater, Patrick M: 1 article (01/2007)|
|4.||Ghosh, Mimi: 1 article (01/2007)|
|5.||Adams, Robert J: 1 article (01/2007)|
|6.||Reinhart, Todd A: 1 article (01/2007)|
|7.||Zink, M Christine: 1 article (01/2007)|
|8.||Pardo, Carlos A: 1 article (01/2007)|
|9.||Queen, Suzanne E: 1 article (01/2007)|
|10.||Jacobs, Amy: 1 article (10/2004)|
|1.||HIV Infections (HIV Infection)
08/10/2001 - "SIV gp41 binds to membranes both in the monomeric and trimeric states: consequences for the neuropathology and inhibition of HIV infection."
09/01/1999 - "Monomer-trimer equilibrium of the ectodomain of SIV gp41: insight into the mechanism of peptide inhibition of HIV infection."
01/01/1995 - "aa 603-622 of SIV gp41, like aa 579-613 of HIV gp41, can stimulate production of antibodies that enhance SIV and HIV infection in vitro. "
01/01/2007 - "To characterize the inflammatory cell population and measure productive viral infection in ganglia, trigeminal ganglia from SIV-infected macaques were immunostained for macrophage or cytotoxic lymphocyte markers and for SIV gp41. "
01/01/1995 - "To demonstrate that antibodies against amino acids (aa) 603-622 of the SIV gp41 transmembrane glycoprotein enhance infection of SIV in vivo. "
|3.||Prion Diseases (Transmissible Spongiform Encephalopathies)
10/01/2004 - "Previously, we have shown by electron microscopy that the extracellular domain of SIV gp41, which is analogous to that of HIV, forms high-molecular weight aggregates in vitro, and we have postulated that such high-molecular weight aggregates are responsible for neurological damage in a manner similar to protein deposition diseases such as Alzheimer's and the prion diseases. "
|4.||Encephalitis (Encephalitis, Rasmussen)
01/01/2002 - "SIV encephalitis was present in 9 of the 10 animals with elevated beta-APP Increases in beta-APP correlated most strongly with levels of SIV gp41 in the brain (p = 0.005), but significant associations with macrophage infiltration and microglial activation (p = 0.04) and infiltration by cytotoxic lymphocytes (p = 0.05) also were identified. "