|1.||Tian, Hui: 2 articles (03/2015 - 01/2012)|
|2.||Wang, Ping: 2 articles (02/2015 - 01/2014)|
|3.||Cai, Hua: 2 articles (02/2015 - 01/2014)|
|4.||Bouhidel, Jalaleddinne Omar: 2 articles (02/2015 - 01/2014)|
|5.||Sahuquillo, Juan: 2 articles (10/2014 - 07/2011)|
|6.||O, Karmin: 2 articles (07/2009 - 10/2008)|
|7.||Xu, Zhibin: 2 articles (07/2009 - 10/2008)|
|8.||Siow, Yaw L: 2 articles (07/2009 - 10/2008)|
|9.||Zhang, De-Fang: 1 article (03/2015)|
|10.||Zheng, Jun-Nian: 1 article (03/2015)|
|1.||Colorectal Neoplasms (Colorectal Cancer)
02/01/2015 - "Experiments with mice deficient in netrin-1 receptor DCC (deleted in colorectal cancer, DCC+/-), or reperfusion with netrin-1/UO126 or netrin-1/PTIO, attenuated the protective effects of netrin-1, implicating intermediate roles of DCC, ERK1/2 and NO. Netrin-1 induced phosphorylation of ERK1/2 and eNOS was abolished in DCC+/-mice. "
03/01/2000 - "In conclusion, this study demonstrates the ability of a combined probe to measure simultaneously regional myocardial PtiO(2)and metabolite concentration during hypoxia. "
10/01/2014 - "The PtiO(2)/PaO(2) ratio is a better indicator than absolute PtiO(2) in detecting tissue hypoxia in mechanically ventilated patients. "
02/01/2003 - "In human astrocytoma U-373 MG cells, hypoxia increased intracellular Ca2+ concentration ([Ca2+]i) by 67.2 +/- 13.1% compared to normoxic controls and this effect was inhibited by L-NAME, PTIO or heparin plus RR. "
01/01/2013 - "The extent of TEMPOL or C-PTIO reduction was similar to the yield of HNO formed upon oxidation by ()OH under anoxia, but not by the metmyoglobin and H(2)O(2) reaction system where both nitroxides catalytically facilitate H(2)O(2) depletion and nitrite accumulation. "
03/01/2000 - "Similarly, myocardial oxygen enrichment, measured by the P O(2)electrode, was negligible when microdialysis probe perfusion flow was 2microl/min. Systemic hypoxia (FiO(2)=0.08) resulted in a significant decrease in PtiO(2)from 30+/-4 to 11+/-2 mmHg, limited increase in coronary blood flow (CBF), and a significant increase in myocardial adenosine and lactate concentrations from 0.34+/-0.05 to 0.98+/-0.06micromol/l and from 0.45+/-0.05 to 0.97+/-0.06 mmol/l respectively (P<0.05). "
01/01/2011 - "Brain infarction occurred in 2 patients; in both cases, there was a decrease in PtiO(2) greater than 80% from basal value, a minimum value of less than 2 mmHg persisting for 2 or more minutes during temporary clipping, and an incomplete recovery of PtiO(2) after definitive clipping. "
01/01/2011 - "The values of risk for brain infarction during temporary arterial occlusion still need further studies, but an incomplete recovery or a persistent fall in PtiO(2) values after definitive clipping should be considered as an indication for verification of the position of the clip."
09/01/2000 - "Mean arterial pressure (MAP), heart rate (HR), Pao(2), and Ptio(2) were measured 30 min before and 0, 60, and 120 min after induction of endotoxemia. "
09/01/2000 - "The objective of this study was to investigate the effects of amrinone on arterial oxygenation (Pao(2)) and tissue oxygenation (Ptio(2)) of jejunal mucosa during endotoxemia. "
01/01/2015 - "NO production in the tumor was changed using ITU, an iNOS inhibitor; c-PTIO, a NO scavenger; and SNP, a NO donor. "
04/10/2003 - "Consequently, lowering intracellular NO levels with either L-NAA or PTIO significantly raised ROS levels, indicating that endogenously generated NO may play a role as a ROS scavenger, thereby protecting exponentially growing tumor spheroids from ROS-induced apoptosis."
03/01/1997 - "In contrast to the effect of L-NNA, constant i.v. infusion of C-PTIO had no effect on tumor or normal tissue blood flow. "
01/01/2012 - "The NO donor, sodium nitroprusside (SNP), down-regulates Bcl-2 S-denitrosylation, attenuates Bcl-2 ubiquitination and subsequently counteracts MDA-7/IL-24 induced cancer cell apoptosis, whereas NO inhibitor 2-(4-carboxyphenyl)-4,4,5,5-tetramethylimidazoline-1-oxy-3-oxide (PTIO) shows the opposite effect. "
03/01/2015 - "We found that NO inhibitor 2-(4-carboxyphenyl)-4,4,5,5-tetramethylimidazoline-1-oxy-3-oxide (PTIO) promoted caspase-3 denitrosylation and caspase-3 cleavage, thereby exacerbating ZD55-IL-24-induced cancer cell apoptosis, whereas NO donor sodium nitroprusside (SNP) showed the opposite effect. "
|1.||U 0126 (UO 126)
|5.||Nitric Oxide (Nitrogen Monoxide)
|6.||Nitric Oxide Synthase (NO Synthase)
|7.||NG-Nitroarginine Methyl Ester (L-NAME)
|8.||2- phenyl- 4,4,5,5- tetramethylimidazoline- 1- oxyl- 3- oxide
|10.||Nitroprusside (Sodium Nitroprusside)
|1.||Surgical Instruments (Clip)