|1.||Yang, Zichao: 1 article (02/2013)|
|2.||Zhang, Shuyan: 1 article (02/2013)|
|3.||Dong, Qi: 1 article (02/2013)|
|4.||Su, Danying: 1 article (02/2013)|
|5.||Liu, Bin: 1 article (02/2013)|
|6.||Zhang, Shuangyan: 1 article (02/2013)|
|7.||Lv, Manhua: 1 article (02/2013)|
|8.||Moldrich, Randal X: 1 article (08/2003)|
|9.||De Sarro, Giovambattista: 1 article (08/2003)|
|10.||Meldrum, Brian S: 1 article (08/2003)|
07/20/2001 - "Another Group III agonist, (RS)-4-phosphonophenyl-glycine (PPG), preferentially activating the mGlu(8) receptor, previously shown to protect against sound-induced seizures in DBA/2 mice and GEP rats, also protects against seizures induced in DBA/2 by 3,5-DHPG (ED(50) 3.7 [2.4-5.7], nmol i.c.v.) and by the Group III antagonist, MSOP (ED(50) 40.2 [21.0-77.0], nmol i.c.v.). "
02/01/1998 - "In conclusion, the in vivo activation of group I mGluRs with the selective agonist 3,5-DHPG produces hyperexcitatory effects that lead to seizures and neuronal damage, these effects being more severe than those observed after infusion of the non-selective agonist 1S,3R-ACPD."
02/01/1998 - "Seizures and neuronal damage induced in the rat by activation of group I metabotropic glutamate receptors with their selective agonist 3,5-dihydroxyphenylglycine."
08/01/1995 - "Interestingly, L-AP4, L-SOP and low doses of L-CCGI also protected against 3,5-DHPG seizures. "
08/01/1995 - "Injections of 3,5-DHPG, 1S,3R-ACPD and L-CCGI produced dose-dependent increases in limbic seizures with a potency order of 3,5-DHPG = 1S,3R-ACPD > L-CCGI. "
10/01/1996 - "When (R,S)-alpha-methyl-4-carboxyphenylglycine was applied prior to high-frequency tetanus and ADA or 3,5-dihydroxyphenylglycine applied 30 min after high-frequency tetanus, or after short-term potentiation decay, elicited no facilitation of long-term potentiation. "
10/01/1996 - "Similar effects were seen with 3,5-dihydroxyphenylglycine applied 25 min after high-frequency tetanus. "
10/01/1996 - "Application of ADA (20 mM/5 microliters) or 3,5-dihydroxyphenylglycine (4mM/5 microliters) 5 min after high-frequency tetanus facilitated short-term potentiation into a long-term potentiation which lasted over 24 h. "
01/01/1998 - "A 10-min application of the group I mGluR agonist 3,5-dihydroxyphenylglycine (DHPG) caused a transient depression of synaptic responses but a significant enhancement of subsequent LTP for both tetanus protocols (45% and 41% LTP, respectively). "
10/01/1996 - "(R,S)-alpha-Methyl-4-carboxyphenylglycine (200 mM/5 microliters), a metabotropic glutamate receptor antagonist, when applied prior to high-frequency tetanus and ADA or 3,5-dihydroxyphenylglycine, completely inhibited this effect. "
|4.||Absence Epilepsy (Absence Seizure)
|1.||Metabotropic Glutamate Receptors (Metabotropic Glutamate Receptor)
|7.||methylserine phosphate (MSOP)