|1.||Mehta, P D: 4 articles (05/2008 - 01/2000)|
|2.||Kang, Da-Yeon: 2 articles (12/2009 - 07/2009)|
|3.||Oh, Byung-Keun: 2 articles (12/2009 - 07/2009)|
|4.||Kim, Sang-Uk: 2 articles (12/2009 - 07/2009)|
|5.||Lee, Jin-Ho: 2 articles (12/2009 - 07/2009)|
|6.||Watson, M D: 2 articles (09/2003 - 04/2000)|
|7.||Emmerling, M R: 2 articles (09/2003 - 04/2000)|
|8.||Mehta, S P: 2 articles (05/2001 - 01/2000)|
|9.||Carrotta, Rita: 1 article (12/2013)|
|10.||Canale, Claudio: 1 article (12/2013)|
|1.||Alzheimer Disease (Alzheimer's Disease)
12/01/2009 - "Numerous studies have indentified that beta-amyloid (1-40) in cerebrospinal fluid as a potential biomarker for Alzheimer's disease. "
07/01/2009 - "Numerous studies have shown that the presence of beta-amyloid (1-40) in cerebrospinal fluid can be used as a potential biomarker for Alzheimer's disease. "
05/06/2008 - "To examine the association between incident Alzheimer disease (AD), and plasma A beta 1-40 and A beta 1-42 levels in normal and mild cognitive impairment (MCI) subjects in a subgroup of participants of the Cardiovascular Health Study Cognition Study. "
05/18/2001 - "Amyloid beta protein 1-40 and 1-42 levels in matched cerebrospinal fluid and plasma from patients with Alzheimer disease."
06/05/1995 - "Beta amyloid 1-40 is a primary protein in plaques found in the brains of patients with Alzheimer's disease. "
|2.||Memory Disorders (Memory Loss)
09/01/2003 - "In conclusion, our study shows that CBH initiates spatial memory loss in aging rats thus mimicking human MCI and also increases A beta 1-40 in the hippocampus. "
12/13/2002 - "Our data indicate that chronic accumulation of beta-amyloid (1-40) into the rat brain reduced activation of PKC, effect that would substantially contribute to the memory deficit found in these animals."
09/01/2003 - "Hippocampal nitric oxide upregulation precedes memory loss and A beta 1-40 accumulation after chronic brain hypoperfusion in rats."
10/18/2002 - "The effects of melatonin and Ginkgo biloba extract on memory loss and choline acetyltransferase activities in the brain of rats infused intracerebroventricularly with beta-amyloid 1-40."
06/15/2000 - "We have studied the toxic effects of both soluble and aggregated species of A beta(1-40) and the mutation A beta(1-40)Glu-->Gln(22), which is the major species deposited in the cerebrovascular blood vessels of victims of hereditary cerebral hemorrhage with amyloidosis, Dutch type. "
04/01/2000 - "In particular, A beta 1-40 carrying the E22Q mutation, as in hereditary cerebral hemorrhage with amyloidosis of the Dutch type (HCHWA-D), is toxic. "
03/01/1998 - "Here we present a kinetic study that indicates laminin to be a potent anti-amyloidosis factor, as it not only inhibited A beta 1-40 fibril aggregation, but also inhibited the aggregation of the Dutch A beta 1-40 variant, a peptide with a higher capacity to aggregate than the wild-type A beta 1-40. "
03/01/1997 - "A beta 1-42 polymerizes more rapidly in vitro  than A beta 1-40 and has been identified as the major component of the brain amyloid deposits [7-9]. "
06/14/1996 - "In this report we show that ACT, a minor protein component of beta-amyloid deposits, is able to inhibit A beta (1-40) aggregation into fibrils, but unable to modulate the toxicity of A beta (1-40) in primary rat hippocampal cell cultures. "
09/01/1992 - "A beta 1-40 preparation from another laboratory was tested that did not form neuronal beta-amyloid deposits and was not neurotoxic. "
11/04/1996 - "Intra-arterial infusion of [125I]A beta 1-40 labels amyloid deposits in the aged primate brain in vivo."
03/01/1996 - "In nontransgenic rat models, intraventricular infusion of A beta 1-40 (alone) generally results in diffuse deposition of A beta with very few focal plaque-like amyloid deposits after a 30-day intraventricular infusion. "
04/01/1998 - "Second, CSF levels of A beta species (CSF-A beta) with different carboxy termini, i.e., A beta X-40 and A beta X-42 (43) as well as A beta 1-40 and A beta 1-42 (43), were measured in patients with AD and in age-matched controls without dementia using ELISA. "
06/30/1998 - "Cerebrospinal fluid samples from a total of 157 subjects consisting of 55 patients with sporadic Alzheimer's disease (AD), 34 normal controls, 23 patients with non-AD dementia, and 45 with other neurological diseases were examined by ELISA of tau, A beta 1-40, and A beta 1-42(43). "
07/01/1998 - "To clarify the alterations of tau, amyloid beta protein (A beta) 1-40 and A beta1-42(43) in the cerebrospinal fluid (CSF) that accompany normal aging and the progression of Alzheimer's disease (AD), CSF samples of 93 AD patients, 32 longitudinal subjects among these 93 AD patients, 33 patients with non-AD dementia, 56 with other neurological diseases, and 54 normal control subjects from three independent institutes were analyzed by sensitive enzyme-linked immunosorbent assays. "
01/01/2000 - "To examine plasma and cerebrospinal fluid (CSF) levels of amyloid beta protein 1-40 (Abeta40) and 1-42 (Abeta42) levels in patients with probable Alzheimer disease (AD) and elderly nondemented control subjects in relation to the apolipoprotein E (Apo E) genotype and dementia severity. "
|6.||Amyloid (Amyloid Fibrils)