|1.||Molgó, Jordi: 2 articles (12/2011 - 04/2005)|
|2.||Foudah, Ahmed I: 1 article (01/2015)|
|3.||Ebrahim, Hassan Y: 1 article (01/2015)|
|4.||El Sayed, Khalid A: 1 article (01/2015)|
|5.||Mohyeldin, Mohamed M: 1 article (01/2015)|
|6.||Ayoub, Nehad M: 1 article (01/2015)|
|7.||Akl, Mohamed R: 1 article (01/2015)|
|8.||Orabi, Khaled Y: 1 article (01/2015)|
|9.||Parys, Jan B: 1 article (12/2011)|
|10.||Ponsaerts, Raf: 1 article (12/2011)|
|1.||Breast Neoplasms (Breast Cancer)
07/01/2009 - "Xestospongin B- or starvation-induced autophagy was inhibited by overexpression of the IP(3)R ligand-binding domain, which coimmunoprecipitated with Beclin 1. These results identify IP(3)R as a new regulator of the Beclin 1 complex that may bridge signals converging on the ER and initial phagophore formation."
12/01/2011 - "Moreover, recombinant Beclin 1 sensitized Ins(1,4,5)P3Rs in 45Ca2+-flux assays, indicating a direct regulation of Ins(1,4,5)P3R activity by Beclin 1. Finally, we found that Ins(1,4,5)P3R-mediated Ca2+ signaling was critical for starvation-induced autophagy stimulation, since the Ca2+ chelator BAPTA-AM as well as the Ins(1,4,5)P3R inhibitor xestospongin B abolished the increase in LC3 lipidation and GFP-LC3-puncta formation. "
04/11/2005 - "It is concluded that xestospongin B is an effective cell-permeant, competitive inhibitor of IP(3) receptors in cultured rat myotubes, isolated myonuclei, and neuroblastoma (NG108-15) cells."
04/11/2005 - "Xestospongin B, a competitive inhibitor of IP3-mediated Ca2+ signalling in cultured rat myotubes, isolated myonuclei, and neuroblastoma (NG108-15) cells."
04/11/2005 - "Xestospongin B, depending on the dose, suppressed bradykinin-induced Ca(2+) signals in neuroblastoma (NG108-15) cells, and also selectively blocked the slow intracellular Ca(2+) signal induced by membrane depolarization with high external K(+) (47 mM) in rat skeletal myotubes. "
|3.||1,2-bis(2-aminophenoxy)ethane N,N,N',N'-tetraacetic acid acetoxymethyl ester