|1.||Wu, Fu-Hui: 1 article (12/2011)|
|2.||Wang, Ai-Yu: 1 article (12/2011)|
|3.||Yeh, Sheng-Hsiung: 1 article (12/2011)|
|4.||Lin, Choun-Sea: 1 article (12/2011)|
|5.||Tasma, I Made: 1 article (01/2009)|
|6.||Sandhu, Devinder: 1 article (01/2009)|
|7.||Bhattacharyya, Madan K: 1 article (01/2009)|
|8.||Frasch, Ryan: 1 article (01/2009)|
|9.||Chae, Lee: 1 article (12/2003)|
|10.||Verica, Joseph A: 1 article (12/2003)|
01/01/2009 - "Pathogenesis-related gene GmPR1 is induced following treatment of soybean plants with the SAR inducer, 2,6-dichloroisonicotinic acid (INA) or infection with the oomycete pathogen, Phytophthora sojae. "
05/01/2003 - "Salicylic acid (SA), benzo-(1,2,3)-thiadiazole-7-carbothioic S-methyl ester (BTH), 2,6-dichloroisonicotinic acid (INA) or EDTA protected sunflower plants against Botrytis infection, that was revealed by a reduction in the number and area of the necrotic lesions in upper leaves after challenge inoculation with the pathogen. "
09/01/2000 - "Abstract Salicylic acid (SA) and its synthetic mimics 2,6-dichloroisonicotinic acid (DCINA) and benzo(1,2,3)thiadiazole-7-carbothioic acid S-methyl ester (BTH), protect barley systemically against powdery mildew (Blumeria graminis f.sp. hordei, Bgh) infection by strengthening plant defence mechanisms that result in effective papillae and host cell death. "
10/01/1998 - "The Arabidopsis PR-1 gene is one of a suite of genes induced co-ordinately during the onset of systemic acquired resistance (SAR), a plant defense pathway triggered by pathogen infection or exogenous application of chemicals such as salicylic acid (SA) and 2,6-dichloroisonicotinic acid (INA). "
12/01/2011 - "The expression level of this gene in multiple shoots of bamboo, which were cultured in vitro, was also upregulated by auxins, cytokinins, pathogen infection, 2,6-dichloroisonicotinic acid (a functional analog of salicylic acid), and hydrogen peroxide. "
|2.||Wounds and Injuries (Trauma)
12/01/2003 - "The expression of WAKL5 and WAKL7 can be induced by wounding stress and by the salicylic acid analog 2,6-dichloroisonicotinic acid in an nonexpressor of pathogenesis-related gene 1-dependent manner, suggesting that they, like some WAK members, are wound inducible and can be defined as pathogenesis-related genes."
|1.||Salicylic Acid (2 Hydroxybenzoic Acid)
|2.||Indoleacetic Acids (Auxin)
|3.||Hydrogen Peroxide (Hydroperoxide)
|4.||Edetic Acid (EDTA)
|7.||S-methyl benzo(1,2,3)thiadiazole-7-carbothioate (acibenzolar-S-methyl)