|1.||Moss, Stephen E: 2 articles (06/2009 - 04/2004)|
|2.||Bailly, Maryse: 2 articles (06/2009 - 04/2004)|
|3.||Hayes, Matthew J: 2 articles (06/2009 - 04/2004)|
|4.||Engelhardt, John F: 2 articles (01/2003 - 12/2002)|
|5.||Fan, Chenguang: 2 articles (01/2003 - 12/2002)|
|6.||Ponnappan, Subramaniam: 1 article (01/2015)|
|7.||Ponnappan, Usha: 1 article (01/2015)|
|8.||Cullen, Sarah: 1 article (01/2015)|
|9.||Geahlen, Robert L: 1 article (10/2013)|
|10.||Yu, Shuai: 1 article (10/2013)|
12/01/2000 - "Pervanadate induces the hyperphosphorylation but not the activation of human heat shock factor 1."
12/01/2000 - "In this study, we evaluated the effects of pervanadate, a tyrosine phosphatase inhibitor, on the regulation and function of heat-shock factor 1 (HSF1) in HeLa cells. "
04/02/2004 - "We show here that macropinosome rocketing can be induced by hyperosmotic shock, either alone or synergistically when combined with phorbol ester or pervanadate. "
12/01/2000 - "Upon heat shock, the pervanadate-induced hyperphosphorylated HSF1 formed DNA-binding trimers and translocated into the nuclear compartment. "
06/01/2009 - "Annexin A2 co-localised with comet-tails induced by pervanadate and hyperosmotic shock in a basophilic cell line, and in an epithelial cell line upon activation of PKC. "
|2.||Melanoma (Melanoma, Malignant)
01/01/2011 - "Cellular stress was induced in mouse proximal tubular epithelial cell line by treating with pervanadate, cisplatin, lipopolysaccharide, glucose or hypoxia followed by reoxygenation. "
03/01/1998 - "The tyrosine phosphatase inhibitor, pervanadate, reversed the hypoxia-induced activation of CPT-I and returned the affinity of cardiac CPT-I for carnitine to control. "
01/01/2015 - "Alternative redox stimuli such as pervanadate or hypoxia/reoxygenation, induce transcription factor NF-κB by phospho-tyrosine-dependent and proteasome-independent mechanisms. "
01/17/2003 - "Tyrosine 42 phosphorylation of I kappa B alpha has been shown to mediate NF kappa B activation following hypoxia/reoxygenation (H/R) or pervanadate treatment. "
12/15/2002 - "To this end, we constructed a recombinant adenoviral vector (Ad.IkappaBalphaAS) expressing antisense IkappaBalpha mRNA that is capable of augmenting NFkappaB activation prior to and following four types of cellular injury [TNF-alpha, UV, hypoxia/reoxygenation (H/R) or pervanadate treatment]. "
|4.||Breast Neoplasms (Breast Cancer)
03/15/1999 - "Cleavage of the HER2 ectodomain is a pervanadate-activable process that is inhibited by the tissue inhibitor of metalloproteases-1 in breast cancer cells."
10/01/2013 - "An examination of the RelA (p65) subunit of NF-κB expressed in MCF7 breast cancer cells indicated that either treatment with pervanadate or stable expression of Syk protected RelA from calpain-mediated proteolysis. "
12/01/2010 - "Pervanadate, a general tyrosine phosphatase inhibitor, revealed marked phosphatase regulation of Stat5 activity in breast cancer cells. "
10/10/2003 - "Tyrosine phosphorylation of PTEN is increased in breast cancer cells treated with pervanadate, suggesting that PTEN contains sites for tyrosine phosphorylation. "
09/09/1999 - "Through its SH2 domain, Grb2 can constitutively associate with transmembranal PTPepsilon in mammary tumors initiated by ras or neu, and can be induced to associate with cytoplasmic PTPepsilon in Jurkat T-cells following stimulation of T-cell receptor signaling by pervanadate. "
05/01/2000 - "Tumor-derived mutated E-cadherin influences beta-catenin localization and increases susceptibility to actin cytoskeletal changes induced by pervanadate."
07/15/2002 - "SSeCKS, a major protein kinase C (PKC) substrate with protein scaffolding and tumor suppressive properties, becomes tyrosine phosphorylated in NIH3T3 and rodent embryo fibroblasts after short-term treatment with epidermal growth factor (EGF), platelet-derived growth factor (PDGF), or fetal calf serum in the presence of pervanadate, but not by treatment with insulin or insulin-like growth factor-1. "
|3.||Protein Tyrosine Phosphatases
|4.||NF-kappa B (NF-kB)
|5.||Transcription Factors (Transcription Factor)
|6.||Proteins (Proteins, Gene)
|9.||NF-kappaB inhibitor alpha